KLF8 involves in TGF-beta-induced EMT and promotes invasion and migration in gastric cancer cells. - Test2 - elhamkhani - TriplyDB

KLF8 involves in TGF-beta-induced EMT and promotes invasion and migration in gastric cancer cells.

KLF8 involves in TGF-beta-induced EMT and promotes invasion and migration in gastric cancer cells.

http://www.wikidata.org/entity/Q39179487

about

Regulation of epithelial-mesenchymal transition by tumor-associated macrophages in cancer Epithelial-mesenchymal transition in gastric cancer Role of cancer-associated fibroblasts in invasion and metastasis of gastric cancer Normal fibroblasts induce E-cadherin loss and increase lymph node metastasis in gastric cancer Long non-coding RNA regulation of epithelial-mesenchymal transition in cancer metastasis Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition Notch signaling is important for epithelial-mesenchymal transition induced by low concentrations of doxorubicin in osteosarcoma cell lines.KLF8 knockdown suppresses proliferation and invasion in human osteosarcoma cells.Tumor-associated macrophages promote Ezrin phosphorylation-mediated epithelial-mesenchymal transition in lung adenocarcinoma through FUT4/LeY up-regulation Epigenetic silencing of DACH1 induces the invasion and metastasis of gastric cancer by activating TGF-β signalling Genistein suppresses FLT4 and inhibits human colorectal cancer metastasis.Krüppel-like factor 8 contributes to hypoxia-induced MDR in gastric cancer cells.Significances of contactin-1 expression in human gastric cancer and knockdown of contactin-1 expression inhibits invasion and metastasis of MKN45 gastric cancer cells.Metformin inhibits gastric cancer cells metastatic traits through suppression of epithelial-mesenchymal transition in a glucose-independent manner.N-methyl-N-nitro-N'-nitrosoguanidine induces the expression of CCR2 in human gastric epithelial cells promoting CCL2-mediated migration.Cadherin-12 enhances proliferation in colorectal cancer cells and increases progression by promoting EMT.Krüppel-like factor 8 activates the transcription of C-X-C cytokine receptor type 4 to promote breast cancer cell invasion, transendothelial migration and metastasis.KLF5 activates microRNA 200 transcription to maintain epithelial characteristics and prevent induced epithelial-mesenchymal transition in epithelial cells.Identification of epithelial stromal interaction 1 as a novel effector downstream of Krüppel-like factor 8 in breast cancer invasion and metastasis Krüppel-like factors in cancer progression: three fingers on the steering wheel.Clinical significance of epithelial-mesenchymal transition.Biomechanics of TGFβ-induced epithelial-mesenchymal transition: implications for fibrosis and cancer Pancreatic cancer stem cells.Expression of Kruppel-like factor 8 and Ki67 in lung adenocarcinoma and prognosis.Epithelial-mesenchymal transition in liver fibrosis HOXB8 promotes tumor metastasis and the epithelial-mesenchymal transition via ZEB2 targets in gastric cancer.Crk-like adapter protein is required for TGF-β-induced AKT and ERK-signaling pathway in epithelial ovarian carcinomas.Gastric cancer cell growth and epithelial-mesenchymal transition are inhibited by γ-secretase inhibitor DAPT.SP and KLF Transcription Factors in Digestive Physiology and Diseases.Pleiotropic Roles of Non-Coding RNAs in TGF-β-Mediated Epithelial-Mesenchymal Transition and Their Functions in Tumor Progression Mesenchymal stem cells promote pancreatic adenocarcinoma cells invasion by transforming growth factor-β1 induced epithelial-mesenchymal transition.Krüppel-like factor 8 induces epithelial-to-mesenchymal transition and promotes invasion of pancreatic cancer cells through transcriptional activation of four and a half LIM-only protein 2 Krüppel-like factor 8 ameliorates Alzheimer's disease by activating β-catenin.The role of glutamate decarboxylase 65 in gastric cancer development, progression, and prognosis.Site-specific fibroblasts regulate site-specific inflammatory niche formation in gastric cancer.

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P2860

KLF8 involves in TGF-beta-induced EMT and promotes invasion and migration in gastric cancer cells.

http://www.wikidata.org/entity/Q39179487

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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type

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KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

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Journal of Cancer Research and Clinical Oncology

P1476

KLF8 involves in TGF-beta-indu ...... ation in gastric cancer cells.

@en

P2093

Changhao Liu

http://www.w3.org/2001/XMLSchema#string

Daiming Fan

http://www.w3.org/2001/XMLSchema#string

Guohong Zhao

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Hongbo Zhang

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Hui Zhang

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Kaichun Wu

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Lili Liu

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Rougang Xie

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Xiao Xiao

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Yafang Wang

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P2860

Human Krüppel-like factor 8: a CACCC-box binding protein that associates with CtBP and represses transcription.

A network of Krüppel-like Factors (Klfs). Klf8 is repressed by Klf3 and activated by Klf1 in vivo

Epithelial-mesenchymal transitions in development and disease

Krüppel-like factor 8 induces epithelial to mesenchymal transition and epithelial cell invasion

KLF8 transcription factor participates in oncogenic transformation

Association between endogenous gene expression and growth regulation induced by TGF-beta1 in human gastric cancer cells

Natural killer cell cytotoxicity and immunosuppressive cytokines (IL-10, TGF-beta1) in patients with gastric cancer.

Requirement of podocalyxin in TGF-beta induced epithelial mesenchymal transition.

The biology of the mammalian Krüppel-like family of transcription factors.

Neuropilin-2 expression promotes TGF-β1-mediated epithelial to mesenchymal transition in colorectal cancer cells.

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6

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