FANCD2 Facilitates Replication through Common Fragile Sites. - Test2 - elhamkhani - TriplyDB

FANCD2 Facilitates Replication through Common Fragile Sites.

FANCD2 Facilitates Replication through Common Fragile Sites.

http://www.wikidata.org/entity/Q39261664

about

Potential biomarkers of DNA replication stress in cancer.Rescue from replication stress during mitosis.Maintenance of genome stability by Fanconi anemia proteins.The "enemies within": regions of the genome that are inherently difficult to replicate.Replication Fork Protection Factors Controlling R-Loop Bypass and Suppression Dormant origins as a built-in safeguard in eukaryotic DNA replication against genome instability and disease development.RNA Binding Proteins and Genome Integrity.The Role of Replication-Associated Repair Factors on R-Loops Close encounters: Moving along bumps, breaks, and bubbles on expanded trinucleotide tracts.Precarious maintenance of simple DNA repeats in eukaryotes.Fragile sites in cancer: more than meets the eye.Assessing the spectrum of germline variation in Fanconi anemia genes among patients with head and neck carcinoma before age 50.Phosphorylated SIRT1 associates with replication origins to prevent excess replication initiation and preserve genomic stability.Replication stress induces accumulation of FANCD2 at central region of large fragile genes.FANCD2 binding identifies conserved fragile sites at large transcribed genes in avian cells.Human THO-Sin3A interaction reveals new mechanisms to prevent R-loops that cause genome instability.Fanconi anemia FANCD2 and FANCI proteins regulate the nuclear dynamics of splicing factors.DNA-RNA hybrids: the risks of DNA breakage during transcription.Replication Stress Shapes a Protective Chromatin Environment across Fragile Genomic Regions.Dormant origins and fork protection mechanisms rescue sister forks arrested by transcription.Extra View: Sirt1 Acts As A Gatekeeper Of Replication Initiation To Preserve Genomic Stability.Pathways for maintenance of telomeres and common fragile sites during DNA replication stress.Human DNA Helicase B as a Candidate for Unwinding Secondary CGG Repeat Structures at the Fragile X Mental Retardation Gene.Parvovirus minute virus of mice interacts with sites of cellular DNA damage to establish and amplify its lytic infection Recent advances in understanding DNA replication: cell type-specific adaptation of the DNA replication program

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P2860

FANCD2 Facilitates Replication through Common Fragile Sites.

http://www.wikidata.org/entity/Q39261664

description

2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh

2016年學術文章

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name

FANCD2 Facilitates Replication through Common Fragile Sites.

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FANCD2 Facilitates Replication through Common Fragile Sites.

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type

label

FANCD2 Facilitates Replication through Common Fragile Sites.

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FANCD2 Facilitates Replication through Common Fragile Sites.

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prefLabel

FANCD2 Facilitates Replication through Common Fragile Sites.

@en

FANCD2 Facilitates Replication through Common Fragile Sites.

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J.MOLCEL.2016.09.017

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FANCD2 Facilitates Replication through Common Fragile Sites

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Carl L Schildkraut

http://www.w3.org/2001/XMLSchema#string

Elizabeth A Vuono

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Jeannine Gerhardt

http://www.w3.org/2001/XMLSchema#string

Niall G Howlett

http://www.w3.org/2001/XMLSchema#string

Nichole Owen

http://www.w3.org/2001/XMLSchema#string

Rebecca A Boisvert

http://www.w3.org/2001/XMLSchema#string

Settapong T Kosiyatrakul

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Susan Olson

http://www.w3.org/2001/XMLSchema#string

Zi Yan

http://www.w3.org/2001/XMLSchema#string

P2860

Frequent dysregulation of the c-maf proto-oncogene at 16q23 by translocation to an Ig locus in multiple myeloma

Epithelial cancer in Fanconi anemia complementation group D2 (Fancd2) knockout mice

Chromosome translocations in multiple myeloma

S-phase-specific interaction of the Fanconi anemia protein, FANCD2, with BRCA1 and RAD51

Interaction of the Fanconi anemia proteins and BRCA1 in a common pathway

Initiation of the breakage-fusion-bridge mechanism through common fragile site activation in human breast cancer cells: the model of PIP gene duplication from a break at FRA7I

Chromosome fragile sites

Ubiquitinated Fancd2 recruits Fan1 to stalled replication forks to prevent genome instability

Biallelic inactivation of BRCA2 in Fanconi anemia

DNA structure and the Werner protein modulate human DNA polymerase delta-dependent replication dynamics within the common fragile site FRA16D.

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388-404

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scholarly article

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10.1016/J.MOLCEL.2016.09.017

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2

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64

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Andrés Aguilera

María L García-Rubio

Advaitha Madireddy

Emilia Herrera-Moyano

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2016-10-01T00:00:00Z

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27768874

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5683400

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