Stability of erp loci during Borrelia burgdorferi infection: recombination is not required for chronic infection of immunocompetent mice.
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Differential binding of host complement inhibitor factor H by Borrelia burgdorferi Erp surface proteins: a possible mechanism underlying the expansive host range of Lyme disease spirochetesSpirochaetal lipoproteins and pathogenesisTemporal expression analysis of the Borrelia burgdorferi paralogous gene family 54 genes BBA64, BBA65, and BBA66 during persistent infection in miceThe relapsing fever spirochete Borrelia hermsii contains multiple, antigen-encoding circular plasmids that are homologous to the cp32 plasmids of Lyme disease spirochetes.Functional characterization of Borrelia spielmanii outer surface proteins that interact with distinct members of the human factor H protein family and with plasminogenBorrelia burgdorferi B31 Erp proteins that are dominant immunoblot antigens of animals infected with isolate B31 are recognized by only a subset of human lyme disease patient sera.A second allele of eppA in Borrelia burgdorferi strain B31 is located on the previously undetected circular plasmid cp9-2Mutation and recombination in the upstream homology box-flanked ospE-related genes of the Lyme disease spirochetes result in the development of new antigenic variants during infection.Molecular and evolutionary characterization of the cp32/18 family of supercoiled plasmids in Borrelia burgdorferi 297.Stability of Borrelia burgdorferi bdr loci in vitro and in vivo.Distinct regulatory pathways control expression of Borrelia burgdorferi infection-associated OspC and Erp surface proteinsChanges in temporal and spatial patterns of outer surface lipoprotein expression generate population heterogeneity and antigenic diversity in the Lyme disease spirochete, Borrelia burgdorferiSimultaneous coexpression of Borrelia burgdorferi Erp proteins occurs through a specific, erp locus-directed regulatory mechanism.Linear and circular plasmid content in Borrelia burgdorferi clinical isolates.Borrelia burgdorferi complement regulator-acquiring surface protein 2 (CspZ) as a serological marker of human Lyme disease.Distribution of cp32 prophages among Lyme disease-causing spirochetes and natural diversity of their lipoprotein-encoding erp lociMolecular characterization of Borrelia burgdorferi erp promoter/operator elements.Borrelia burgdorferi erp (ospE-related) gene sequences remain stable during mammalian infection.Analysis of the OspE determinants involved in binding of factor H and OspE-targeting antibodies elicited during Borrelia burgdorferi infection in mice.Natural selection promotes antigenic evolvability.
P2860
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P2860
Stability of erp loci during Borrelia burgdorferi infection: recombination is not required for chronic infection of immunocompetent mice.
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1999 nî lūn-bûn
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1999年の論文
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1999年論文
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1999年論文
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1999年論文
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1999年論文
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1999年論文
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1999年论文
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1999年论文
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1999年论文
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name
Stability of erp loci during B ...... ction of immunocompetent mice.
@en
Stability of erp loci during B ...... ction of immunocompetent mice.
@nl
type
label
Stability of erp loci during B ...... ction of immunocompetent mice.
@en
Stability of erp loci during B ...... ction of immunocompetent mice.
@nl
prefLabel
Stability of erp loci during B ...... ction of immunocompetent mice.
@en
Stability of erp loci during B ...... ction of immunocompetent mice.
@nl
P2093
P2860
P1476
Stability of erp loci during B ...... ection of immunocompetent mice
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P2093
P2860
P304
P407
P577
1999-06-01T00:00:00Z