Pulmonary epithelial CCR3 promotes LPS-induced lung inflammation by mediating release of IL-8.
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Chemokine receptors and their therapeutic opportunities in diseased lung: far beyond leukocyte traffickingAn extraribosomal function of ribosomal protein L13a in macrophages resolves inflammationComparison of methods to reduce myocardial 18F-FDG uptake in mice: calcium channel blockers versus high-fat diets.Host-Mycobacterium avium subsp. paratuberculosis interactome reveals a novel iron assimilation mechanism linked to nitric oxide stress during early infection.An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infectionShp2 plays an important role in acute cigarette smoke-mediated lung inflammationAdh enhances Actinobacillus pleuropneumoniae pathogenicity by binding to OR5M11 and activating p38 which induces apoptosis of PAMs and IL-8 releaseMesenchymal stem cells in acute lung injury: are they ready for translational medicine?Instilled air promotes lipopolysaccharide-induced acute lung injury.The mercurial nature of neutrophils: still an enigma in ARDS?Poly-L-Arginine Acts Synergistically with LPS to Promote the Release of IL-6 and IL-8 via p38/ERK Signaling Pathways in NCI-H292 Cells.Maternal molecular hydrogen treatment attenuates lipopolysaccharide-induced rat fetal lung injury.Protective effects of intratracheally administered quercetin on lipopolysaccharide-induced acute lung injury.Tight junctions in pulmonary epithelia during lung inflammation.Influence of surface charge on the potential toxicity of PLGA nanoparticles towards Calu-3 cells.Anacardic acid, a histone acetyltransferase inhibitor, modulates LPS-induced IL-8 expression in a human alveolar epithelial cell line A549.Comparative study of trans-oral and trans-tracheal intratracheal instillations in a murine model of acute lung injury.Regulatory mechanisms of interleukin-8 production induced by tumour necrosis factor-α in human hepatocellular carcinoma cells.Dual-Drug Containing Core-Shell Nanoparticles for Lung Cancer TherapyAttenuation of pulmonary ACE2 activity impairs inactivation of des-Arg9 bradykinin/BKB1R axis and facilitates LPS-induced neutrophil infiltration.Juglanin suppresses fibrosis and inflammation response caused by LPS in acute lung injury.
P2860
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P2860
Pulmonary epithelial CCR3 promotes LPS-induced lung inflammation by mediating release of IL-8.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@en
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@nl
type
label
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@en
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@nl
prefLabel
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@en
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@nl
P2093
P2860
P356
P1476
Pulmonary epithelial CCR3 prom ...... by mediating release of IL-8.
@en
P2093
Chunling Dong
Chunxue Bai
Guifang Wang
Huiru Zheng
Xiangdong Wang
P2860
P304
P356
10.1002/JCP.22577
P577
2011-09-01T00:00:00Z