Loss of CCR2 signaling alters leukocyte recruitment and exacerbates γ-herpesvirus-induced pneumonitis and fibrosis following bone marrow transplantation.

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IL-17 in the lung: The Good, the Bad, and the Ugly.Bone marrow transplant-induced alterations in Notch signaling promote pathologic Th17 responses to γ-herpesvirus infection.Fibrocytes are increased in lung and peripheral blood of patients with idiopathic pulmonary fibrosis.

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P2860

Loss of CCR2 signaling alters leukocyte recruitment and exacerbates γ-herpesvirus-induced pneumonitis and fibrosis following bone marrow transplantation.

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2016 nî lūn-bûn

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2016年の論文

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Loss of CCR2 signaling alters ...... g bone marrow transplantation.

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Carol A Wilke

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David N O'Dwyer

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Megan C Procario

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Stephen J Gurczynski

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P2860

CCR2 deficiency promotes exacerbated chronic erosive neutrophil-dominated chikungunya virus arthritis

Mechanisms of alveolar epithelial injury, repair, and fibrosis

Idiopathic pneumonia syndrome after bone marrow transplantation: the role of pre-transplant radiation conditioning and local cytokine dysregulation in promoting lung inflammation and fibrosis

Eosinophils and T lymphocytes possess distinct roles in bleomycin-induced lung injury and fibrosis

Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.

Arginase-1-expressing macrophages suppress Th2 cytokine-driven inflammation and fibrosis

Lung parenchyma-derived IL-6 promotes IL-17A-dependent acute lung injury after allogeneic stem cell transplantation

γ-Herpes virus-68, but not Pseudomonas aeruginosa or influenza A (H1N1), exacerbates established murine lung fibrosis

CCR2-mediated recruitment of fibrocytes to the alveolar space after fibrotic injury.

Fibrocytes are not an essential source of type I collagen during lung fibrosis.

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scholarly article

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10.1152/AJPLUNG.00193.2016

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Bethany B Moore

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Loss of CCR2 signaling alters leukocyte recruitment and exacerbates γ-herpesvirus-induced pneumonitis and fibrosis following bone marrow transplantation.

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P2860

P2860

Loss of CCR2 signaling alters leukocyte recruitment and exacerbates γ-herpesvirus-induced pneumonitis and fibrosis following bone marrow transplantation.

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