Opposite functions of HIF-α isoforms in VEGF induction by TGF-β1 under non-hypoxic conditions.
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Multiple functions of the RNA-binding protein HuR in cancer progression, treatment responses and prognosisHypoxia-inducing factors as master regulators of stemness properties and altered metabolism of cancer- and metastasis-initiating cellsHDAC6 deacetylase activity is required for hypoxia-induced invadopodia formation and cell invasion.The acetate/ACSS2 switch regulates HIF-2 stress signaling in the tumor cell microenvironment.Immunohistochemical assessment of intrinsic and extrinsic markers of hypoxia in reproductive tissue: differential expression of HIF1α and HIF2α in rat oviduct and endometrium.Frequent gene products and molecular pathways altered in prostate cancer- and metastasis-initiating cells and their progenies and novel promising multitargeted therapies.The role of hypoxia-inducible factor-2 in digestive system cancers.Cross regulation between hypoxia-inducible transcription factor-1α (HIF-1α) and transforming growth factor (TGF)-ß1 mediates nickel oxide nanoparticles (NiONPs)-induced pulmonary fibrosisHypoxia-inducible factor prolyl-hydroxylase-2 mediates transforming growth factor beta 1-induced epithelial-mesenchymal transition in renal tubular cells.Inhibition of ARNT severely compromises endothelial cell viability and function in response to moderate hypoxia.PPARδ promotes oncogenic redirection of TGF-β1 signaling through the activation of the ABCA1-Cav1 pathway.Electromagnetic field-induced converse cell growth during a long-term observation.MicroRNA-145 inhibits the growth, invasion, metastasis and angiogenesis of neuroblastoma cells through targeting hypoxia-inducible factor 2 alpha.Hypoxia induces calpain activity and degrades SMAD2 to attenuate TGFβ signaling in macrophages.Vascular endothelial growth factor A, secreted in response to transforming growth factor-β1 under hypoxic conditions, induces autocrine effects on migration of prostate cancer cells.Radio-induced apoptosis of peripheral blood CD8 T lymphocytes is a novel prognostic factor for survival in cervical carcinoma patients.Endothelial HIF-2α Contributes to Severe Pulmonary Hypertension by Inducing Endothelial-to-Mesenchymal Transition.A microfluidic oxygen gradient demonstrates differential activation of the hypoxia-regulated transcription factors HIF-1α and HIF-2α.Augmentation of hypoxia-inducible factor-1-alpha in reinfused blood cells enhances diabetic ischemic wound closure in mice.
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P2860
Opposite functions of HIF-α isoforms in VEGF induction by TGF-β1 under non-hypoxic conditions.
description
2010 nî lūn-bûn
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2010年の論文
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2010年論文
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2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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2010年论文
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2010年论文
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name
Opposite functions of HIF-α is ...... under non-hypoxic conditions.
@en
Opposite functions of HIF-α is ...... under non-hypoxic conditions.
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Opposite functions of HIF-α is ...... under non-hypoxic conditions.
@en
Opposite functions of HIF-α is ...... under non-hypoxic conditions.
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Opposite functions of HIF-α is ...... under non-hypoxic conditions.
@en
Opposite functions of HIF-α is ...... under non-hypoxic conditions.
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P2093
P2860
P356
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P1476
Opposite functions of HIF-α is ...... under non-hypoxic conditions.
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P2093
P2860
P2888
P304
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10.1038/ONC.2010.498
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P577
2010-11-08T00:00:00Z