Aberrantly expressed AURKC enhances the transformation and tumourigenicity of epithelial cells. - Test2 - elhamkhani - TriplyDB

Aberrantly expressed AURKC enhances the transformation and tumourigenicity of epithelial cells.

Aberrantly expressed AURKC enhances the transformation and tumourigenicity of epithelial cells.

http://www.wikidata.org/entity/Q39734794

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Functions of Aurora kinase C in meiosis and cancer Possible Role of Aurora-C in Meiosis Aurora-C Interactions with Survivin and INCENP Reveal Shared and Distinct Features Compared with Aurora-B Chromosome Passenger Protein Complex A new aurora in anaplastic thyroid cancer therapy.Overexpression of active Aurora-C kinase results in cell transformation and tumour formation.The aurora kinases in cell cycle and leukemia Ran GTPase-activating protein 1 is a therapeutic target in diffuse large B-cell lymphoma.Analysis of the interaction between Zinc finger protein 179 (Znf179) and promyelocytic leukemia zinc finger (Plzf).Overexpression of Aurora-C interferes with the spindle checkpoint by promoting the degradation of Aurora-B.Transcriptome analysis of the cancer/testis genes, DAZ1, AURKC, and TEX101, in breast tumors and six breast cancer cell lines.Haspin kinase regulates microtubule-organizing center clustering and stability through Aurora kinase C in mouse oocytes.Regulation of AURKC expression by CpG island methylation in human cancer cells.Single nucleotide polymorphisms rs911160 in AURKA and rs2289590 in AURKB mitotic checkpoint genes contribute to gastric cancer susceptibility.Association of the AURKA and AURKC gene polymorphisms with an increased risk of gastric cancer.Potential new biomarkers for squamous carcinoma of the uterine cervix.The functional diversity of Aurora kinases: a comprehensive review Identification of Key Pathways and Genes in Anaplastic Thyroid Carcinoma via Integrated Bioinformatics Analysis

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Aberrantly expressed AURKC enhances the transformation and tumourigenicity of epithelial cells.

http://www.wikidata.org/entity/Q39734794

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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2011年學術文章

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name

Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

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Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

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type

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Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

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Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

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Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

@en

Aberrantly expressed AURKC enh ...... igenicity of epithelial cells.

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The Journal of Pathology

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Aberrantly expressed AURKC enh ...... rigenicity of epithelial cells

@en

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Bo-Wen Lin

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Chin-Kai Chuang

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Chung-Ta Lee

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Jen-Hui Tsou

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Jenq-Chang Lee

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Ju-Ming Wang

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Kung-Chao Chang

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Liang-Yi Hung

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Meng-Ru Shen

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Pey-Yi Chang-Liao

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P2860

A novel cervical cancer suppressor 3 (CCS-3) interacts with the BTB domain of PLZF and inhibits the cell growth by inducing apoptosis

Regulation of Hoxb2 by APL-associated PLZF protein

The promyelocytic leukemia zinc finger (PLZF) protein binds DNA in a high molecular weight complex associated with cdc2 kinase

Evolutionary relationships of Aurora kinases: implications for model organism studies and the development of anti-cancer drugs

Overexpression of an Aurora-C kinase-deficient mutant disrupts the Aurora-B/INCENP complex and induces polyploidy

Histone deacetylase associated with mSin3A mediates repression by the acute promyelocytic leukemia-associated PLZF protein

Direct association with inner centromere protein (INCENP) activates the novel chromosomal passenger protein, Aurora-C

Protein kinase profile of sperm and eggs: cloning and characterization of two novel testis-specific protein kinases (AIE1, AIE2) related to yeast and fly chromosome segregation regulators

Raf kinase inhibitory protein regulates aurora B kinase and the spindle checkpoint

Plzf is required in adult male germ cells for stem cell self-renewal

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243-254

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scholarly article

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10.1002/PATH.2934

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2

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225

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2011-06-27T00:00:00Z

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51255097

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21710690

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