New host nuclear functions are not required for the modifications of the parasitophorous vacuole of Toxoplasma. - Test2 - elhamkhani - TriplyDB

New host nuclear functions are not required for the modifications of the parasitophorous vacuole of Toxoplasma.

New host nuclear functions are not required for the modifications of the parasitophorous vacuole of Toxoplasma.

http://www.wikidata.org/entity/Q40059981

about

Host cell manipulation by the human pathogen Toxoplasma gondii Host Organelle Hijackers: a similar modus operandi for Toxoplasma gondii and Chlamydia trachomatis: co-infection model as a tool to investigate pathogenesis A novel co-infection model with Toxoplasma and Chlamydia trachomatis highlights the importance of host cell manipulation for nutrient scavenging Fierce competition between Toxoplasma and Chlamydia for host cell structures in dually infected cells.Sphingolipid synthesis and scavenging in the intracellular apicomplexan parasite, Toxoplasma gondii Toxoplasma gondii salvages sphingolipids from the host Golgi through the rerouting of selected Rab vesicles to the parasitophorous vacuole.Membrane traffic and synaptic cross-talk during host cell entry by Trypanosoma cruzi.Trans-sialidase stimulates eat me response from epithelial cells.The parasite Toxoplasma sequesters diverse Rab host vesicles within an intravacuolar network.Hostile intruder: Toxoplasma holds host organelles captive.

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P2860

New host nuclear functions are not required for the modifications of the parasitophorous vacuole of Toxoplasma.

http://www.wikidata.org/entity/Q40059981

description

2007 nî lūn-bûn

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2007年の論文

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2007年论文

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New host nuclear functions are ...... phorous vacuole of Toxoplasma.

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New host nuclear functions are ...... phorous vacuole of Toxoplasma.

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New host nuclear functions are ...... phorous vacuole of Toxoplasma.

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J.1462-5822.2007.01061.X

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Cellular Microbiology

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New host nuclear functions are ...... ophorous vacuole of Toxoplasma

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Isabelle Coppens

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Nazneen Bano

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P2860

Ionophore-resistant mutants of Toxoplasma gondii reveal host cell permeabilization as an early event in egress.

Daughter cell assembly in the protozoan parasite Toxoplasma gondii

Lytic cycle of Toxoplasma gondii.

Ricin. Mechanisms of cytotoxicity.

Cell cycle-dependent changes in microtubule dynamics in living cells expressing green fluorescent protein-alpha tubulin.

Toxoplasma gondii targets a protein phosphatase 2C to the nuclei of infected host cells.

Host but not parasite cholesterol controls Toxoplasma cell entry by modulating organelle discharge.

Role of the centrosome in organizing the interphase microtubule array: properties of cytoplasts containing or lacking centrosomes

The protozoan parasite Toxoplasma gondii targets proteins to dense granules and the vacuolar space using both conserved and unusual mechanisms.

Toxoplasma gondii exploits host low-density lipoprotein receptor-mediated endocytosis for cholesterol acquisition

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465-476

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scholarly article

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10.1111/J.1462-5822.2007.01061.X

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2

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10

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17970763

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