about
Proliferating cell nuclear antigen acts as a cytoplasmic platform controlling human neutrophil survivalInduction and regulatory function of miR-9 in human monocytes and neutrophils exposed to proinflammatory signalsAntibodies against human cytomegalovirus in the pathogenesis of systemic sclerosis: a gene array approachClinical and immunological data of nine patients with chronic mucocutaneous candidiasis diseaseAn additional piece in the puzzle of neutrophil-derived IL-1β: the NLRP3 inflammasome.Identification of TLR4 as the receptor that recognizes Shiga toxins in human neutrophils.IL-10-induced microRNA-187 negatively regulates TNF-α, IL-6, and IL-12p40 production in TLR4-stimulated monocytes.Soluble TNF-like cytokine (TL1A) production by immune complexes stimulated monocytes in rheumatoid arthritis.TL1A/DR3 axis involvement in the inflammatory cytokine network during pulmonary sarcoidosis.RelB activation in anti-inflammatory decidual endothelial cells: a master plan to avoid pregnancy failure?Proteinase 3 on apoptotic cells disrupts immune silencing in autoimmune vasculitisIFNα enhances the production of IL-6 by human neutrophils activated via TLR8.Understanding the molecular mechanisms of the multifaceted IL-10-mediated anti-inflammatory response: lessons from neutrophils.Cytoplasmic receptors recognizing nucleic acids and mediating immune functions in neutrophils.On the potential involvement of CD11d in co-stimulating the production of interferon-γ by natural killer cells upon interaction with neutrophils via intercellular adhesion molecule-3.Reduction of CRKL expression in patients with partial DiGeorge syndrome is associated with impairment of T-cell functions.Epigenetic regulation of neutrophil development and function.Neutrophil-Expressed p21/waf1 Favors Inflammation Resolution in Pseudomonas aeruginosa Infection.The MyD88-independent pathway is not mobilized in human neutrophils stimulated via TLR4.Endogenously produced TNF-α contributes to the expression of CXCL10/IP-10 in IFN-λ3-activated plasmacytoid dendritic cells.Clinical heterogeneity of dominant chronic mucocutaneous candidiasis disease: presenting as treatment-resistant candidiasis and chronic lung disease.IL-10 disrupts the Brd4-docking sites to inhibit LPS-induced CXCL8 and TNF-α expression in monocytes: Implications for chronic obstructive pulmonary disease.Chromatin remodelling and autocrine TNFα are required for optimal interleukin-6 expression in activated human neutrophils.Fast and accurate quantitative analysis of cytokine gene expression in human neutrophils.Severe impairment of IFN-γ and IFN-α responses in cells of a patient with a novel STAT1 splicing mutation.Endothelial cells' activation and apoptosis induced by a subset of antibodies against human cytomegalovirus: relevance to the pathogenesis of atherosclerosisIFN-β expression is directly activated in human neutrophils transfected with plasmid DNA and is further increased via TLR-4-mediated signaling.Proinflammatory mediators elicit secretion of the intracellular B-lymphocyte stimulator pool (BLyS) that is stored in activated neutrophils: implications for inflammatory diseases.Activation of an immunoregulatory and antiviral gene expression program in poly(I:C)-transfected human neutrophils.Fast and accurate quantitative analysis of cytokine gene expression in human neutrophils by reverse transcription real-time PCR.Interferon-activated neutrophils store a TNF-related apoptosis-inducing ligand (TRAIL/Apo-2 ligand) intracellular pool that is readily mobilizable following exposure to proinflammatory mediators.Cutting edge: An inactive chromatin configuration at the IL-10 locus in human neutrophils.Neutrophils promote 6-sulfo LacNAc+ dendritic cell (slanDC) survival.Circulating neutrophils of septic patients constitutively express IL-10R1 and are promptly responsive to IL-10.Failure to detect production of IL-10 by activated human neutrophilsSH2-domain mutations in STAT3 in hyper-IgE syndrome patients result in impairment of IL-10 functionUncovering an IL-10-dependent NF-κB recruitment to the IL-1ra promoter that is impaired in STAT3 functionally defective patientsslanDCs selectively accumulate in carcinoma-draining lymph nodes and marginate metastatic cellsToll-Like Receptor-3-Activated Human Mesenchymal Stromal Cells Significantly Prolong the Survival and Function of NeutrophilsEvidence for a cross-talk between human neutrophils and Th17 cells
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Nicola Tamassia
@ast
Nicola Tamassia
@en
Nicola Tamassia
@es
Nicola Tamassia
@nl
Nicola Tamassia
@sl
type
label
Nicola Tamassia
@ast
Nicola Tamassia
@en
Nicola Tamassia
@es
Nicola Tamassia
@nl
Nicola Tamassia
@sl
prefLabel
Nicola Tamassia
@ast
Nicola Tamassia
@en
Nicola Tamassia
@es
Nicola Tamassia
@nl
Nicola Tamassia
@sl
P1053
J-8621-2016
P106
P21
P31
P3829
P496
0000-0003-2990-3003