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Evidence that 5-HT2A receptors in the hypothalamic paraventricular nucleus mediate neuroendocrine responses to (-)DOIA region-specific increase in Galphaq and Galpha11 proteins in brains of rats during cocaine withdrawalPhospholipase C, Ca2+, and calmodulin signaling are required for 5-HT2A receptor-mediated transamidation of Rac1 by transglutaminase6-hydroxydopamine-induced lesions of dopaminergic neurons alter the function of postsynaptic cholinergic neurons without changing cytoskeletal proteins.Neuroprotective effects of calmodulin peptide 76-121aa: disruption of calmodulin binding to mutant huntingtin.Activation of the JAK-STAT pathway by olanzapine is necessary for desensitization of serotonin2A receptor-stimulated phospholipase C signaling in rat frontal cortex but not serotonin2A receptor-stimulated hormone releaseNeuroendocrine evidence that (S)-2-(chloro-5-fluoro-indol- l-yl)-1-methylethylamine fumarate (Ro 60-0175) is not a selective 5-hydroxytryptamine(2C) receptor agonist.Extra-nuclear estrogen receptor GPR30 regulates serotonin function in rat hypothalamus.GPER1 stimulation alters posttranslational modification of RGSz1 and induces desensitization of 5-HT1A receptor signaling in the rat hypothalamusEstradiol induces partial desensitization of serotonin 1A receptor signaling in the paraventricular nucleus of the hypothalamus and alters expression and interaction of RGSZ1 and GαzCocaine-mediated supersensitivity of 5-HT2A receptors in hypothalamic paraventricular nucleus is a withdrawal-induced phenomenon.GPR30 is necessary for estradiol-induced desensitization of 5-HT1A receptor signaling in the paraventricular nucleus of the rat hypothalamus.Use of a force-sensing automated open field apparatus in a longitudinal study of multiple behavioral deficits in CAG140 Huntington's disease model miceChronic olanzapine activates the Stat3 signal transduction pathway and alters expression of components of the 5-HT2A receptor signaling system in rat frontal cortex.Estradiol accelerates the effects of fluoxetine on serotonin 1A receptor signalingTransglutaminase activation in neurodegenerative diseases.Neuronal disorders: studies of animal models and human diseases.Evidence that G(z)-proteins couple to hypothalamic 5-HT(1A) receptors in vivo.Receptor-stimulated transamidation induces activation of Rac1 and Cdc42 and the regulation of dendritic spines.Activation of the JAK-STAT pathway is necessary for desensitization of 5-HT2A receptor-stimulated phospholipase C signalling by olanzapine, clozapine and MDL 100907.Protective effects of interrupting the binding of calmodulin to mutant huntingtin.Agonist induced-phosphorylation of Galpha11 protein reduces coupling to 5-HT2A receptors.Olanzapine increases RGS7 protein expression via stimulation of the Janus tyrosine kinase-signal transducer and activator of transcription signaling cascade.Phosphorylation of Galpha11 protein contributes to agonist-induced desensitization of 5-HT2A receptor signaling.Mutant huntingtin protein: a substrate for transglutaminase 1, 2, and 3.Calmodulin regulates transglutaminase 2 cross-linking of huntingtin.Transglutaminase cross-links in intranuclear inclusions in Huntington disease.Postnatal increases in neurofilament gene expression correlate with the radial growth of axons.Aluminum neurotoxicity: altered expression of cytoskeletal genes.Neuronal gene expression in aluminum-induced neurofibrillary pathology: an in situ hybridization study.Striatal expression of a calmodulin fragment improved motor function, weight loss, and neuropathology in the R6/2 mouse model of Huntington's disease.Alternative splicing of amino-terminal Tau mRNA in rat spinal cord during development and following axonal injury.Characterization of the functional heterologous desensitization of hypothalamic 5-HT(1A) receptors after 5-HT(2A) receptor activation.Brain and serum levels of choline and lecithin resulting from long-term administration.Transglutaminase bonds in neurofibrillary tangles and paired helical filament tau early in Alzheimer's disease.Treatment of cycling female rats with fluoxetine induces desensitization of hypothalamic 5-HT(1A) receptors with no change in 5-HT(2A) receptors.Differences in regional and subcellular localization of G(q/11) and RGS4 protein levels in Alzheimer's disease: correlation with muscarinic M1 receptor binding parameters.Ovariectomy-induced increases in hypothalamic serotonin-1A receptor function in rats are prevented by estradiol.Transglutaminase activity, protein, and mRNA expression are increased in progressive supranuclear palsy.Chronic fluoxetine differentially affects 5-hydroxytryptamine (2A) receptor signaling in frontal cortex, oxytocin- and corticotropin-releasing factor-containing neurons in rat paraventricular nucleus.
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P50
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հետազոտող
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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Nancy A Muma
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P106
P21
P31
P496
0000-0001-8272-3138