about
CXCR7 influences leukocyte entry into the CNS parenchyma by controlling abluminal CXCL12 abundance during autoimmunityThe AP-1 transcription factor Batf controls T(H)17 differentiationBTLA is a lymphocyte inhibitory receptor with similarities to CTLA-4 and PD-1Dysmyelination revealed through MRI as increased radial (but unchanged axial) diffusion of water.Detecting axon damage in spinal cord from a mouse model of multiple sclerosis.Batf3-dependent CD11b(low/-) peripheral dendritic cells are GM-CSF-independent and are not required for Th cell priming after subcutaneous immunization.Immune system dysfunction and autoimmune disease in mice lacking Emk (Par-1) protein kinase.Natural killer and lymphokine-activated killer cells require granzyme B for the rapid induction of apoptosis in susceptible target cells.Lymphocyte-mediated cytotoxicity.A tumor necrosis factor receptor 1-dependent conversation between central nervous system-specific T cells and the central nervous system is required for inflammatory infiltration of the spinal cord.Regional CNS responses to IFN-gamma determine lesion localization patterns during EAE pathogenesisAxonal injury detected by in vivo diffusion tensor imaging correlates with neurological disability in a mouse model of multiple sclerosis.Encephalitogenic T-cells increase numbers of CNS T-cells regardless of antigen specificity by both increasing T-cell entry and preventing egress.T-cell trafficking competence is required for CNS invasion.Activation-induced death of mature T cells in the regulation of immune responses.Role of the immune response in interstitial cystitis.Partial signaling by cytokines: cytokine regulation of cell cycle and Fas-dependent, activation-induced death in CD4+ subsets.Detachment and lysis of adherent target cells by CD4+ T cell clones involve multiple effector mechanisms.TNFR1-dependent VCAM-1 expression by astrocytes exposes the CNS to destructive inflammation.Region-specific regulation of inflammation and pathogenesis in experimental autoimmune encephalomyelitis.Defining antigen-dependent stages of T cell migration from the blood to the central nervous system parenchyma.Toward accurate diagnosis of white matter pathology using diffusion tensor imaging.Maternal-fetal tolerance is maintained despite transgene-driven trophoblast expression of MHC class I, and defects in Fas and its ligand.Cytotoxic lymphocytes require granzyme B for the rapid induction of DNA fragmentation and apoptosis in allogeneic target cells.Genetic control of cross-reactive cytotoxic T-lymphocyte responses to a BALB/c tumorApoptotic death of lymphocytes in murine acquired immunodeficiency syndrome: involvement of Fas-Fas ligand interactionSeparation of CD4+ functional responses by peptide dose in Th1 and Th2 subsets expressing the same transgenic antigen receptorGenetic control of pathogenic mechanisms in autoimmune demyelinating diseaseQuantitation of the cell surface level of Ld resulting in positive versus negative selection of the 2C transgenic T cell receptor in vivo
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description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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type
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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John H. Russell
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P106
P1153
7404208600
P21
P31
P496
0000-0002-3680-9587