Specific killing of multiple myeloma cells by (-)-epigallocatechin-3-gallate extracted from green tea: biologic activity and therapeutic implications.
about
Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.Apoptosis and autophagy induction as mechanism of cancer prevention by naturally occurring dietary agentsGreen tea polyphenol sensingInteraction of green tea polyphenol epigallocatechin-3-gallate with sunitinib: potential risk of diminished sunitinib bioavailabilityMetabolic profiling-based data-mining for an effective chemical combination to induce apoptosis of cancer cells.Herbs in hemato-oncological care: an evidence-based review of data on efficacy, safety, and drug interactions.PDE3 inhibitor and EGCG combination treatment suppress cancer stem cell properties in pancreatic ductal adenocarcinoma.Multiple functions of the 37/67-kd laminin receptor make it a suitable target for novel cancer gene therapyGreen tea polyphenol EGCG sensing motif on the 67-kDa laminin receptorExtraribosomal functions associated with the C terminus of the 37/67 kDa laminin receptor are required for maintaining cell viability.(-)-Epigallocatechin-3-gallate (EGCG) sensitizes melanoma cells to interferon induced growth inhibition in a mouse model of human melanomaAnti-tumor activity and signaling events triggered by the isothiocyanates, sulforaphane and phenethyl isothiocyanate, in multiple myelomaPhytochemicals in cancer prevention and therapy: truth or dare?Small molecule-sensing strategy and techniques for understanding the functionality of green tea.Impact of Procyanidins from Different Berries on Caspase 8 Activation in Colon Cancer.Complementary and alternative medicines in prostate cancer: from bench to bedside?Genomic evolution in Barrett's adenocarcinoma cells: critical roles of elevated hsRAD51, homologous recombination and Alu sequences in the genome.Epigallocatechin-3-O-gallate up-regulates microRNA-let-7b expression by activating 67-kDa laminin receptor signaling in melanoma cells.67-kDa laminin receptor increases cGMP to induce cancer-selective apoptosisDysfunctional homologous recombination mediates genomic instability and progression in myeloma.Preclinical development of the green tea catechin, epigallocatechin gallate, as an HIV-1 therapy.Perspectives for cancer prevention with natural compoundsApoptosis by dietary agents for prevention and treatment of cancer.Phase I trial of daily oral Polyphenon E in patients with asymptomatic Rai stage 0 to II chronic lymphocytic leukemia.Green tea and prostate cancer: from bench to clinic.Targeting homologous recombination and telomerase in Barrett's adenocarcinoma: impact on telomere maintenance, genomic instability and tumor growth.Chemoprevention of head and neck cancer with green tea polyphenolsGuidelines for supportive care in multiple myeloma 2011.Epigallocatechin-3-gallate (EGCG) for clinical trials: more pitfalls than promises?The TP73 complex network: ready for clinical translation in cancer?Role of Sphingolipids and Metabolizing Enzymes in Hematological Malignancies.Green Tea and Bone Marrow Transplantation: From Antioxidant Activity to Enzymatic and Multidrug-resistance Modulation.Green tea polyphenol epigallocatechin-O-gallate induces cell death by acid sphingomyelinase activation in chronic myeloid leukemia cells.67 kDa laminin receptor (67LR) in normal and neoplastic hematopoietic cells: is its targeting a feasible approach?Targeting RET to induce medullary thyroid cancer cell apoptosis: an antagonistic interplay between PI3K/Akt and p38MAPK/caspase-8 pathways.Natural Compounds As Modulators of Non-apoptotic Cell Death in Cancer Cells.Oxygen partial pressure modulates 67-kDa laminin receptor expression, leading to altered activity of the green tea polyphenol, EGCG.Targeting PI3K and RAD51 in Barrett's adenocarcinoma: impact on DNA damage checkpoints, expression profile and tumor growth.Green tea polyphenol EGCG induces lipid-raft clustering and apoptotic cell death by activating protein kinase Cδ and acid sphingomyelinase through a 67 kDa laminin receptor in multiple myeloma cells.Toxicogenomics of nanoparticulate delivery of etoposide: potential impact on nanotechnology in retinoblastoma therapy.
P2860
Q26827810-EC211D41-0B80-4E1A-9727-9D23B84F1BBDQ26866257-209CB17B-4DB2-4031-A0E2-2A6DBEB64374Q28307754-0338E273-3460-4D2F-B50E-85CA450E4E48Q29012696-46CE59FD-C4EE-414B-A88F-509E4F016B5CQ30919523-AB0561B9-7D54-4EDF-86F7-7E1BE95F4EB9Q33596382-84163AC5-5076-4285-8EAF-EBCB743C6CA5Q33689649-451068EC-BA50-40AA-9116-3A082E46A84EQ33730451-2307D8B5-7C7E-48E9-A1CE-55860E8BF844Q34292725-EE0562A4-EF4B-4CBD-A056-DC61F46F3BCDQ34483179-56E98733-0F79-4AE6-8B55-5501FE24DA0EQ34530415-0E9D095F-6990-4E0C-8503-32C3A7BAEAFCQ35143496-42C3C524-4DEE-46D8-BA3F-897982A4F361Q35155749-25B34720-5A46-4C59-88A4-B8656C0E03E6Q35538578-695AFD38-DDDD-4A10-BE7C-6923084649EDQ35769006-1726F1AB-39DB-4B3D-AF0C-90B513509D0BQ36051522-6E6C97D1-FEB8-4FDF-916E-5307A6ADC9BDQ36120783-64F0888E-66BE-4ED6-B9E4-B78E255B8F0CQ36454002-1F4A6036-CEC7-49FE-A20E-5E0CFF2601D5Q36580431-BA8C8E01-FCA7-4707-91BD-DE01F67B2A34Q37119930-21F66930-F1AF-44CE-BC94-CF46AC3AA66EQ37150917-3D068EEB-4F51-4976-AA11-05F3AA02F824Q37212228-A473091C-BE12-46EE-9835-4B02BEA921F7Q37240356-19084717-1FD7-4F9B-A871-2C4328909E90Q37306304-DD5C5F5D-EE04-4B62-811F-21C796DF1544Q37308282-301B2474-8EAC-49F6-9F77-4A14830E2206Q37614123-A806DBA0-F22C-44DC-9216-1630F3C57F3EQ37776083-D0B5ADE8-279F-4FBB-9BC7-79F6FA88ED07Q37868637-41D103CA-BE34-4E09-ADEE-A1C643188586Q37948040-E862B0EE-5CC7-413A-8AB6-1E5EC6B4F3B1Q38126476-CEBB8C69-0BB3-4F1A-BE08-70470FC3C611Q38495744-D413B5D7-E09C-4994-8008-A5D621BF35C9Q38517370-68123011-0E02-4F04-A713-F586F677C1A7Q38857686-99814CE9-24F4-461F-BC49-19C1A0F2C6FAQ39023088-2F357B43-DDFB-469D-A5A3-C1CB6B67201EQ39208774-94C31096-69CB-4CC8-98B6-B6BC4A84D1F7Q39214221-39F87244-FC6F-4AE3-BDD0-7C3252B8050BQ39299460-32222926-1A88-4BB5-B9C6-613FE5D1A51CQ39383614-418BAA67-7D07-494E-8F5B-89E908E4B038Q39410289-56907732-6275-4009-BE11-65C5AE428C11Q39614066-8E7DEE22-73FD-42C9-AE3A-9CCB97E25A3C
P2860
Specific killing of multiple myeloma cells by (-)-epigallocatechin-3-gallate extracted from green tea: biologic activity and therapeutic implications.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Specific killing of multiple m ...... and therapeutic implications.
@en
type
label
Specific killing of multiple m ...... and therapeutic implications.
@en
prefLabel
Specific killing of multiple m ...... and therapeutic implications.
@en
P2093
P2860
P1433
P1476
Specific killing of multiple m ...... and therapeutic implications.
@en
P2093
Hemanta Koley
Mariateresa Fulciniti
Masood A Shammas
Nikhil C Munshi
Paola Neri
Raj K Goyal
Ramesh B Batchu
Rao Prabhala
Robert C Bertheau
Steven P Treon
P2860
P304
P356
10.1182/BLOOD-2006-05-022814
P407
P577
2006-06-29T00:00:00Z