Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and A-dependent mechanisms in rats and mice. - Test2 - elhamkhani - TriplyDB

Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and A-dependent mechanisms in rats and mice.

Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and A-dependent mechanisms in rats and mice.

http://www.wikidata.org/entity/Q40263672

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Brachystemma calycinum D. Don Effectively Reduces the Locomotor Disability in Dogs with Naturally Occurring Osteoarthritis: A Randomized Placebo-Controlled Trial Moderate ethanol ingestion and cardiovascular protection: from epidemiologic associations to cellular mechanisms Proteinase-activated receptor-2 mediated inhibition of TNFalpha-stimulated JNK activation - A novel paradigm for G(q/11) linked GPCRs TRP channels in neurogastroenterology: opportunities for therapeutic intervention The substrates and binding partners of protein kinase Cepsilon The emergence of proteinase-activated receptor-2 as a novel target for the treatment of inflammation-related CNS disorders The role of Transient Receptor Potential Vanilloid 1 (TRPV1) channels in pancreatitis Treatment of Cancer Pain by Targeting Cytokines Biologic mechanisms of oral cancer pain and implications for clinical therapy Nanotechnology-based drug delivery systems for treatment of oral cancer: a review New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Development and evaluation of small peptidomimetic ligands to protease-activated receptor-2 (PAR2) through the use of lipid tethering Hypersensitivity Induced by Activation of Spinal Cord PAR2 Receptors Is Partially Mediated by TRPV1 Receptors Proteinase-activated receptor-4 (PAR4) activation leads to sensitization of rat joint primary afferents via a bradykinin B2 receptor-dependent mechanism The multiple pathways for itch and their interactions with pain Post-synaptic density perturbs insulin-induced Kv1.3 channel modulation via a clustering mechanism involving the SH3 domain.Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors.Transient receptor potential ankyrin-1 has a major role in mediating visceral pain in mice.Identifying the Ion Channels Responsible for Signaling Gastro-Intestinal Based Pain Serine proteases and protease-activated receptor 2-dependent allodynia: a novel cancer pain pathway.[EXPRESS] Ion channels and neuronal hyperexcitability in chemotherapy-induced peripheral neuropathy; cause and effect?Mechanism of cancer pain.Serine proteases and protease-activated receptor 2 mediate the proinflammatory and algesic actions of diverse stimulants.Mast cells: versatile gatekeepers of pain.PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.The active Zot domain (aa 288-293) increases ZO-1 and myosin 1C serine/threonine phosphorylation, alters interaction between ZO-1 and its binding partners, and induces tight junction disassembly through proteinase activated receptor 2 activation TRPV1 activation is required for hypertonicity-stimulated inflammatory cytokine release in human corneal epithelial cells.The protease-activated receptor-2-specific agonists 2-aminothiazol-4-yl-LIGRL-NH2 and 6-aminonicotinyl-LIGRL-NH2 stimulate multiple signaling pathways to induce physiological responses in vitro and in vivo TRPV4-mediated calcium influx into human bronchial epithelia upon exposure to diesel exhaust particles.The distinct roles of two GPCRs, MrgprC11 and PAR2, in itch and hyperalgesia.Nociceptor beta II, delta, and epsilon isoforms of PKC differentially mediate paclitaxel-induced spontaneous and evoked pain.Whiplash-like facet joint loading initiates glutamatergic responses in the DRG and spinal cord associated with behavioral hypersensitivity.Protease-activated receptor 2 activation is sufficient to induce the transition to a chronic pain state New insights into protease-activated receptor 4 signaling pathways in the pathogenesis of inflammation and neuropathic pain: a literature review Protease-activated receptors (PARs)--biology and role in cancer invasion and metastasis.P2Y1 Receptor Activation of the TRPV4 Ion Channel Enhances Purinergic Signaling in Satellite Glial Cells.Protease-activated receptor 2 (PAR2) protein and transient receptor potential vanilloid 4 (TRPV4) protein coupling is required for sustained inflammatory signaling.Inflammatory mediators modulating the transient receptor potential vanilloid 1 receptor: therapeutic targets to treat inflammatory and neuropathic pain.Potentiation of the transient receptor potential vanilloid 1 channel contributes to pruritogenesis in a rat model of liver disease.Evidence for the role of neurogenic inflammation components in trypsin-elicited scratching behaviour in mice.

P2860

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P2860

Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and A-dependent mechanisms in rats and mice.

http://www.wikidata.org/entity/Q40263672

description

2006 nî lūn-bûn

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

Protease-activated receptor 2 ...... t mechanisms in rats and mice.

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Protease-activated receptor 2 ...... t mechanisms in rats and mice.

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Protease-activated receptor 2 ...... t mechanisms in rats and mice.

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JPHYSIOL.2006.111534

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The Journal of Physiology

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Protease-activated receptor 2 ...... t mechanisms in rats and mice.

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Carlos Barajas-Lopez

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Eileen F Grady

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Francisco Bautista

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Graeme S Cottrell

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Kevin Chapman

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Lorna Divino

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Nigel W Bunnett

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Rustum Karanjia

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Silvia Amadesi

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Stephen Vanner

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P2860

beta-arrestin-dependent endocytosis of proteinase-activated receptor 2 is required for intracellular targeting of activated ERK1/2

c-Cbl mediates ubiquitination, degradation, and down-regulation of human protease-activated receptor 2

The capsaicin receptor: a heat-activated ion channel in the pain pathway

Induction of intestinal inflammation in mouse by activation of proteinase-activated receptor-2

Differential regulation of endothelial exocytosis of P-selectin and von Willebrand factor by protease-activated receptors and cAMP

Induction of vanilloid receptor channel activity by protein kinase C

Vanilloid receptor-1 is essential for inflammatory thermal hyperalgesia

Regulated exocytosis contributes to protein kinase C potentiation of vanilloid receptor activity

Protein kinase C activation potentiates gating of the vanilloid receptor VR1 by capsaicin, protons, heat and anandamide

The protease-activated receptor-2 agonist induces gastric mucus secretion and mucosal cytoprotection

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10.1113/JPHYSIOL.2006.111534

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Pt 2

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575

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Nathalie Vergnolle

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2006-06-22T00:00:00Z

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6990429

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16793902

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1819458

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