Circularization of the herpes simplex virus type 1 genome upon lytic infection
about
Keeping it quiet: chromatin control of gammaherpesvirus latencyThe molecular basis of herpes simplex virus latencyStructure of the herpes simplex virus 1 genome: manipulation of nicks and gaps can abrogate infectivity and alter the cellular DNA damage response.Inhibitors of nucleotidyltransferase superfamily enzymes suppress herpes simplex virus replicationRecombination promoted by DNA viruses: phage λ to herpes simplex virus.Varicella zoster virus latency.Evidence that the immediate-early gene product ICP4 is necessary for the genome of the herpes simplex virus type 1 ICP4 deletion mutant strain d120 to circularize in infected cellsGenome wide nucleosome mapping for HSV-1 shows nucleosomes are deposited at preferred positions during lytic infection.Inhibition of IKKα by BAY61-3606 Reveals IKKα-Dependent Histone H3 Phosphorylation in Human Cytomegalovirus Infected CellsCathepsin B mediates cleavage of herpes simplex virus type 1 origin binding protein (OBP) to yield OBPC-1, and cleavage is dependent upon viral DNA replication.Visualizing the replicating HSV-1 virus using STED super-resolution microscopy.High-Throughput Small Interfering RNA Screening Identifies Phosphatidylinositol 3-Kinase Class II Alpha as Important for Production of Human Cytomegalovirus VirionsReplication and recombination of herpes simplex virus DNAViral vectors for gene delivery to the central nervous system.Recent gene therapy advancements for neurological diseases.Dynamic and nucleolin-dependent localization of human cytomegalovirus UL84 to the periphery of viral replication compartments and nucleoli.Coordinated leading and lagging strand DNA synthesis by using the herpes simplex virus 1 replication complex and minicircle DNA templatesNucleolin associates with the human cytomegalovirus DNA polymerase accessory subunit UL44 and is necessary for efficient viral replication.Recent advances in the study of Kaposi's sarcoma-associated herpesvirus replication and pathogenesis.HSV-I and the cellular DNA damage response.An Intrinsically Disordered Region of the DNA Repair Protein Nbs1 Is a Species-Specific Barrier to Herpes Simplex Virus 1 in Primates.H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication.A mutation deleting sequences encoding the amino terminus of human cytomegalovirus UL84 impairs interaction with UL44 and capsid localizationStimulation of homology-directed repair at I-SceI-induced DNA breaks during the permissive life cycle of human cytomegalovirus.Association of human cytomegalovirus proteins IRS1 and TRS1 with the viral DNA polymerase accessory subunit UL44.Analysis of herpes simplex virus type 1 DNA packaging signal mutations in the context of the viral genome.Analysis of the association of the human cytomegalovirus DNA polymerase subunit UL44 with the viral DNA replication factor UL84.Initiation of lytic DNA replication in Epstein-Barr virus: search for a common family mechanism.The UL15 protein of herpes simplex virus type 1 is necessary for the localization of the UL28 and UL33 proteins to viral DNA replication centres.Duck enteritis virus (DEV) UL54 protein, a novel partner, interacts with DEV UL24 protein.The Telomeric Response to Viral Infection.Knockdown of DNA ligase IV/XRCC4 by RNA interference inhibits herpes simplex virus type I DNA replication.Human cytomegalovirus disrupts both ataxia telangiectasia mutated protein (ATM)- and ATM-Rad3-related kinase-mediated DNA damage responses during lytic infection.Upstream-binding factor is sequestered into herpes simplex virus type 1 replication compartments.ICP0 gene expression is a herpes simplex virus type 1 apoptotic triggerPersistence of cyprinid herpesvirus 3 in infected cultured carp cells.The pre-NH(2)-terminal domain of the herpes simplex virus 1 DNA polymerase catalytic subunit is required for efficient viral replication.Herpes simplex virus type 1 C-terminal variants of the origin binding protein (OBP), OBPC-1 and OBPC-2, cooperatively regulate viral DNA levels in vitro, and OBPC-2 affects mortality in mice.Stepwise evolution of the herpes simplex virus origin binding protein and origin of replicationPNKP knockdown by RNA interference inhibits herpes simplex virus-1 replication in astrocytes.
P2860
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P2860
Circularization of the herpes simplex virus type 1 genome upon lytic infection
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Circularization of the herpes simplex virus type 1 genome upon lytic infection
@en
type
label
Circularization of the herpes simplex virus type 1 genome upon lytic infection
@en
prefLabel
Circularization of the herpes simplex virus type 1 genome upon lytic infection
@en
P2860
P1433
P1476
Circularization of the herpes simplex virus type 1 genome upon lytic infection
@en
P2093
Blair L Strang
Nigel D Stow
P2860
P304
12487-12494
P356
10.1128/JVI.79.19.12487-12494.2005
P407
P577
2005-10-01T00:00:00Z