Positive control of proliferation by the cyclic AMP cascade: an oncogenic mechanism of hyper-functional adenoma.

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Positive control of proliferation by the cyclic AMP cascade: an oncogenic mechanism of hyper-functional adenoma.

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1995 nî lūn-bûn

@nan

1995年の論文

@ja

1995年論文

@yue

1995年論文

@zh-hant

1995年論文

@zh-hk

1995年論文

@zh-mo

1995年論文

@zh-tw

1995年论文

@wuu

1995年论文

@zh

1995年论文

@zh-cn

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Positive control of proliferat ...... m of hyper-functional adenoma.

@en

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Item

label

Positive control of proliferat ...... m of hyper-functional adenoma.

@en

prefLabel

Positive control of proliferat ...... m of hyper-functional adenoma.

@en

P1476

Positive control of proliferat ...... m of hyper-functional adenoma.

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P2093

C Ledent

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C Maenhaut

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F Lamy

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I Pirson

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J Parma

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J Van Saude

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M Parmentier

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M Taton

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P Roger

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V Pohl

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P2860

Cloning and functional characterization of a human A1 adenosine receptor

Cloning, sequencing and expression of the human thyrotropin (TSH) receptor: evidence for binding of autoantibodies

Tissue-specific expression and methylation of a thyroglobulin-chloramphenicol acetyltransferase fusion gene in transgenic mice.

Unlike thyrotropin, thyroid-stimulating antibodies do not activate phospholipase C in human thyroid slices.

Activation of the cyclic AMP cascade as an oncogenic mechanism: the thyroid example.

The cyclic AMP-mediated stimulation of cell proliferation.

Somatic mutations in the thyrotropin receptor gene cause hyperfunctioning thyroid adenomas.

Pathogenesis of autonomous thyroid nodules: in vitro study of iodine and adenosine 3',5'-monophosphate metabolism.

RDC8 codes for an adenosine A2 receptor with physiological constitutive activity.

P2888

bf03349717

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120-122

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scholarly article

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10.1007/BF03349717

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1995-02-01T00:00:00Z

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1027463948

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7629377

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