Acute negative inotropic effects of homocysteine are mediated via the endothelium. - Test2 - elhamkhani - TriplyDB

Acute negative inotropic effects of homocysteine are mediated via the endothelium.

Acute negative inotropic effects of homocysteine are mediated via the endothelium.

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Impairment of endothelial-myocardial interaction increases the susceptibility of cardiomyocytes to ischemia/reperfusion injury.Relationship between dietary folate intake and plasma monocyte chemoattractant protein-1 and interleukin-8 in heart failure patients A review of homocysteine and heart failure.Homocysteine, left ventricular dysfunction and coronary artery disease: is there a link?Homocysteine, brain natriuretic peptide and chronic heart failure: a critical review.The effects of the modulation of NMDA receptors by homocysteine thiolactone and dizocilpine on cardiodynamics and oxidative stress in isolated rat heart.Phosphodiesterase type-2 and NO-dependent S-nitrosylation mediate the cardioinhibition of the antihypertensive catestatin.PPAR-gamma agonists decrease hyperhomcysteinemia and cardiac dysfunction: new hope for ailing diabetic hearts?The effects of homocysteine-related compounds on cardiac contractility, coronary flow, and oxidative stress markers in isolated rat heart.Prognostic value of homocysteine and highly sensitive cardiac troponin T in children with acute heart failure.

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Acute negative inotropic effects of homocysteine are mediated via the endothelium.

http://www.wikidata.org/entity/Q40518673

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2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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2004年论文

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Acute negative inotropic effects of homocysteine are mediated via the endothelium.

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Acute negative inotropic effects of homocysteine are mediated via the endothelium.

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Acute negative inotropic effects of homocysteine are mediated via the endothelium.

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AJPHEART.01042.2003

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American Journal of Physiology - Heart and Circulatory Physiology

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Acute negative inotropic effects of homocysteine are mediated via the endothelium.

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P2093

Jacob Joseph

http://www.w3.org/2001/XMLSchema#string

Nawal Shekhawat

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Richard H Kennedy

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Richard Owings

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P2860

Endothelial dysfunction in a murine model of mild hyperhomocyst(e)inemia

Inhibition of K+ currents by homocysteine in rat ventricular myocytes.

Total homocysteine and cardiovascular disease.

Overexpression of cellular glutathione peroxidase rescues homocyst(e)ine-induced endothelial dysfunction

Homocysteine impairs the nitric oxide synthase pathway: role of asymmetric dimethylarginine.

Adverse vascular effects of homocysteine are modulated by endothelium-derived relaxing factor and related oxides of nitrogen

The oxidant stress of hyperhomocyst(e)inemia

Homocysteine reduces smooth muscle [Ca2+]i and constrictor responses of isolated arterioles.

Decreased rate of coronary restenosis after lowering of plasma homocysteine levels.

Determination of different species of homocysteine in human plasma by high-performance liquid chromatography with ultraviolet detection.

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H812-7

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scholarly article

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10.1152/AJPHEART.01042.2003

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2

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287

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2004-04-08T00:00:00Z

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