Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
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Animal models for assessment of infection and inflammation: contributions to elucidating the pathophysiology of sudden infant death syndromeSerotonin in the solitary tract nucleus shortens the laryngeal chemoreflex in anaesthetized neonatal rats.An adenosine A(2A) agonist injected in the nucleus of the solitary tract prolongs the laryngeal chemoreflex by a GABAergic mechanism in decerebrate pigletsTRPV1 channels in the nucleus of the solitary tract mediate thermal prolongation of the LCR in decerebrate pigletsOptogenetic perturbation of preBötzinger complex inhibitory neurons modulates respiratory patternQuantification of rat supraglottic laryngeal sensation threshold.Vanilloids selectively sensitize thermal glutamate release from TRPV1 expressing solitary tract afferentsThe unsilent majority-TRPV1 drives "spontaneous" transmission of unmyelinated primary afferents within cardiorespiratory NTSLaryngeal reflex apnea in neonates: effects of CO2 and the complex influence of hypoxiaAn adenosine A(2A) antagonist injected in the NTS reverses thermal prolongation of the LCR in decerebrate piglets.Impact of rhythmic oral activity on the timing of muscle activation in the swallow of the decerebrate pig.Gestational cigarette smoke exposure and hyperthermic enhancement of laryngeal chemoreflex in rat pups.Potential Central Nervous System Involvement in Sudden Unexpected Infant Deaths and the Sudden Infant Death Syndrome.Interleukin-1β and interleukin-6 enhance thermal prolongation of the LCR in decerebrate piglets.Focal warming in the nucleus of the solitary tract prolongs the laryngeal chemoreflex in decerebrate piglets.Influence of age, body temperature, GABAA receptor inhibition and caffeine on the Hering-Breuer inflation reflex in unanesthetized rat pups.Peptide and lipid modulation of glutamatergic afferent synaptic transmission in the solitary tract nucleus.Breath-to-breath hypercapnic response in neonatal rats: temperature dependency of the chemoreflexes and potential implications for breathing stability.Activation of serotonergic neurons in the medullary caudal raphe shortens the laryngeal chemoreflex in anaesthetized neonatal rats.Unilateral microdialysis of gabazine in the dorsal medulla reverses thermal prolongation of the laryngeal chemoreflex in decerebrate piglets.
P2860
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P2860
Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
@en
type
label
Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
@en
prefLabel
Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
@en
P2093
P2860
P1476
Elevated body temperature enhances the laryngeal chemoreflex in decerebrate piglets.
@en
P2093
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00906.2004
P407
P577
2004-11-12T00:00:00Z