Estrogen receptor activation at serine 305 is sufficient to upregulate cyclin D1 in breast cancer cells.
about
CRIPak, a novel endogenous Pak1 inhibitorIntegration of the extranuclear and nuclear actions of estrogenGPR30: A G protein-coupled receptor for estrogenp21-activated kinase signaling in breast cancerMolecular pathways: targeting p21-activated kinase 1 signaling in cancer--opportunities, challenges, and limitationsPAK as a therapeutic target in gastric cancerInhibition of the Rho GTPase, Rac1, decreases estrogen receptor levels and is a novel therapeutic strategy in breast cancer.Pathways to tamoxifen resistance.A hypersensitive estrogen receptor alpha mutation that alters dynamic protein interactionsp21-Activated Kinases 1, 2 and 4 in Endometrial Cancers: Effects on Clinical Outcomes and Cell Proliferation.Growth factor-induced resistance to tamoxifen is associated with a mutation of estrogen receptor alpha and its phosphorylation at serine 305.Association between the estrogen receptor alpha A908G mutation and outcomes in invasive breast cancer.Elevated expression of mitogen-activated protein kinase phosphatase 3 in breast tumors: a mechanism of tamoxifen resistancePregnane X Receptor-Humanized Mice Recapitulate Gender Differences in Ethanol Metabolism but Not HepatotoxicityTyrosyl phosphorylated serine-threonine kinase PAK1 is a novel regulator of prolactin-dependent breast cancer cell motility and invasion.A Derivative of Differentiation-Inducing Factor-3 Inhibits PAK1 Activity and Breast Cancer Cell Proliferation.Derailed estrogen signaling and breast cancer: an authentic couple.High Expression of Cyclin D1 and p21 in N-Nitroso-N-Methylurea-Induced Breast Cancer in Wistar Albino Female Rats.PAK1 translocates into nucleus in response to prolactin but not to estrogen.Crosstalk between the estrogen receptor and the HER tyrosine kinase receptor family: molecular mechanism and clinical implications for endocrine therapy resistance.Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance.Synergistic Activation of ERα by Estrogen and Prolactin in Breast Cancer Cells Requires Tyrosyl Phosphorylation of PAK1.Pak1, adjuvant tamoxifen therapy, and breast cancer recurrence risk in a Danish population-based study.Evolving pathway-driven biomarkers in breast cancer.Type I receptor tyrosine kinases as predictive or prognostic markers in early breast cancer.Signal integration through blending, bolstering and bifurcating of intracellular information.Phosphorylation of the hinge domain of the nuclear hormone receptor LRH-1 stimulates transactivation.
P2860
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P2860
Estrogen receptor activation at serine 305 is sufficient to upregulate cyclin D1 in breast cancer cells.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Estrogen receptor activation a ...... lin D1 in breast cancer cells.
@en
type
label
Estrogen receptor activation a ...... lin D1 in breast cancer cells.
@en
prefLabel
Estrogen receptor activation a ...... lin D1 in breast cancer cells.
@en
P2093
P2860
P1433
P1476
Estrogen receptor activation a ...... clin D1 in breast cancer cells
@en
P2093
Christopher J Barnes
Rakesh Kumar
Seetharaman Balasenthil
P2860
P304
P356
10.1016/J.FEBSLET.2004.04.071
P407
P577
2004-06-01T00:00:00Z