Interaction between chronically HIV-infected promonocytic cells and human umbilical vein endothelial cells: role of proinflammatory cytokines and chemokines in viral expression modulation.
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Oral infectious diseases: a potential risk factor for HIV virus recrudescence?Primary human mammary epithelial cells endocytose HIV-1 and facilitate viral infection of CD4+ T lymphocytes.Endothelial cells enhance human immunodeficiency virus type 1 replication in macrophages through a C/EBP-dependent mechanism.Acceleration of R5 HIV replication by polymorphonuclear neutrophils in cultures of macrophages.HIV-1 envelope glycoprotein 120 increases intercellular adhesion molecule-1 expression by human endothelial cells.Equine Herpesvirus Type 1 Enhances Viral Replication in CD172a+ Monocytic Cells upon Adhesion to Endothelial CellsSarpogrelate inhibits the expression of ICAM-1 and monocyte-endothelial adhesion induced by high glucose in human endothelial cells.Adhesion of Epstein-Barr virus-positive natural killer cell lines to cultured endothelial cells stimulated with inflammatory cytokines.
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P2860
Interaction between chronically HIV-infected promonocytic cells and human umbilical vein endothelial cells: role of proinflammatory cytokines and chemokines in viral expression modulation.
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2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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name
Interaction between chronicall ...... n viral expression modulation.
@en
type
label
Interaction between chronicall ...... n viral expression modulation.
@en
prefLabel
Interaction between chronicall ...... n viral expression modulation.
@en
P2093
P2860
P50
P1476
Interaction between chronicall ...... in viral expression modulation
@en
P2093
P2860
P304
P356
10.1046/J.1365-2249.2000.01186.X
P577
2000-04-01T00:00:00Z