Long-term cigarette smoke exposure inhibits histone deacetylase 2 expression and enhances the nuclear factor-κB activation in skeletal muscle of mice.
about
Histone deacetylase 2 (HDAC2) attenuates lipopolysaccharide (LPS)-induced inflammation by regulating PAI-1 expression.Genetic ablation of histone deacetylase 2 leads to lung cellular senescence and lymphoid follicle formation in COPD/emphysema.Electroacupuncture: A Feasible Sirt1 Promoter Which Modulates Metainflammation in Diet-Induced Obesity Rats
P2860
Long-term cigarette smoke exposure inhibits histone deacetylase 2 expression and enhances the nuclear factor-κB activation in skeletal muscle of mice.
description
2017 nî lūn-bûn
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2017年の論文
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2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Long-term cigarette smoke expo ...... on in skeletal muscle of mice.
@en
type
label
Long-term cigarette smoke expo ...... on in skeletal muscle of mice.
@en
prefLabel
Long-term cigarette smoke expo ...... on in skeletal muscle of mice.
@en
P2093
P2860
P356
P1433
P1476
Long-term cigarette smoke expo ...... on in skeletal muscle of mice.
@en
P2093
Dongmei Huang
Honglin Luo
Qiuli Liang
Xiaoning Zhong
Zhiying Ma
P2860
P304
56726-56736
P356
10.18632/ONCOTARGET.18089
P407
P577
2017-05-23T00:00:00Z