Increased global transcription activity as a mechanism of replication stress in cancer
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Risks at the DNA Replication Fork: Effects upon Carcinogenesis and Tumor HeterogeneitySomatic Genetic Variation in Solid Pseudopapillary Tumor of the Pancreas by Whole Exome Sequencing.Lineage-Specific Genes Are Prominent DNA Damage Hotspots during Leukemic Transformation of B Cell Precursors.Enhanced dependency of KRAS-mutant colorectal cancer cells on RAD51-dependent homologous recombination repair identified from genetic interactions in Saccharomyces cerevisiae.Emerging Roles for Ciz1 in Cell Cycle Regulation and as a Driver of Tumorigenesis.Effects of Replication and Transcription on DNA Structure-Related Genetic InstabilityReplication Fork Protection Factors Controlling R-Loop Bypass and SuppressionThe Role of the Transcriptional Response to DNA Replication Stress.Regulation of DNA Replication through Natural Impediments in the Eukaryotic Genome.Fragile sites in cancer: more than meets the eye.MDM2 Antagonists Counteract Drug-Induced DNA Damage.Oncogene-Induced Replication Stress Drives Genome Instability and Tumorigenesis.Around and beyond 53BP1 Nuclear Bodies.PARP1 Trapping and DNA Replication Stress Enhance Radiosensitization with Combined WEE1 and PARP Inhibitors.Dormant origins and fork protection mechanisms rescue sister forks arrested by transcription.Transcribing malignancy: transcription-associated genomic instability in cancer.Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.Chromatin conformation regulates the coordination between DNA replication and transcription.Diverse roles of RAD18 and Y-family DNA polymerases in tumorigenesis.RNA/DNA Hybrid Interactome Identifies DXH9 as a Molecular Player in Transcriptional Termination and R-Loop-Associated DNA Damage.Developmental and cancer-associated plasticity of DNA replication preferentially targets GC-poor, lowly expressed and late-replicating regionsIntragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress
P2860
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P2860
Increased global transcription activity as a mechanism of replication stress in cancer
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Increased global transcription activity as a mechanism of replication stress in cancer
@en
type
label
Increased global transcription activity as a mechanism of replication stress in cancer
@en
prefLabel
Increased global transcription activity as a mechanism of replication stress in cancer
@en
P2093
P2860
P50
P356
P1476
Increased global transcription activity as a mechanism of replication stress in cancer
@en
P2093
Lara Marques Silva
Lisa Folkes
Natalia Gromak
Sarah Irmscher
P2860
P2888
P356
10.1038/NCOMMS13087
P407
P577
2016-10-11T00:00:00Z