LRRK2 Antisense Oligonucleotides Ameliorate α-Synuclein Inclusion Formation in a Parkinson's Disease Mouse Model.
about
Critical appraisal of pathology transmission in the α-synuclein fibril model of Lewy body disorders.α-Synuclein fibril-induced paradoxical structural and functional defects in hippocampal neurons.LRRK2 activity does not dramatically alter α-synuclein pathology in primary neurons.Cellular and Molecular Basis of Neurodegeneration in Parkinson Disease.A new hypothesis for Parkinson's disease pathogenesis: GTPase-p38 MAPK signaling and autophagy as convergence points of etiology and genomics
P2860
LRRK2 Antisense Oligonucleotides Ameliorate α-Synuclein Inclusion Formation in a Parkinson's Disease Mouse Model.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
LRRK2 Antisense Oligonucleotid ...... rkinson's Disease Mouse Model.
@en
type
label
LRRK2 Antisense Oligonucleotid ...... rkinson's Disease Mouse Model.
@en
prefLabel
LRRK2 Antisense Oligonucleotid ...... rkinson's Disease Mouse Model.
@en
P2093
P2860
P1476
LRRK2 Antisense Oligonucleotid ...... rkinson's Disease Mouse Model.
@en
P2093
Andreas Weihofen
Andrew B West
Aneeza Kim
Eric E Swayze
Hien Tran Zhao
Holly B Kordasiewicz
Karli Ikeda-Lee
Laura A Volpicelli-Daley
Neena John
Vedad Delic
P2860
P304
P356
10.1016/J.OMTN.2017.08.002
P577
2017-08-10T00:00:00Z