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Identification of a Novel Regulatory Mechanism of Nutrient Transport Controlled by TORC1-Npr1-Amu1/Par32Systematic mutational analysis of the intracellular regions of yeast Gap1 permease.Stress conditions promote yeast Gap1 permease ubiquitylation and down-regulation via the arrestin-like Bul and Aly proteins.Wnt/β-Catenin Signaling Regulates the Expression of the Ammonium Permease Gene RHBG in Human Cancer Cells.Internal amino acids promote Gap1 permease ubiquitylation via TORC1/Npr1/14-3-3-dependent control of the Bul arrestin-like adaptors.Nitrogen catabolite repressible GAP1 promoter, a new tool for efficient recombinant protein production in S. cerevisiae.The metabolic waste ammonium regulates mTORC2 and mTORC1 signaling.Substrate-induced ubiquitylation and endocytosis of yeast amino acid permeases.The MicroRNA miR-210 Is Expressed by Cancer Cells but Also by the Tumor Microenvironment in Triple-Negative Breast Cancer.Silencing of casein kinase 1 delta reduces migration and metastasis of triple negative breast cancer cells
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description
hulumtues
@sq
researcher
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wetenschapper
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հետազոտող
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name
Ahmad Merhi
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Ahmad Merhi
@en
Ahmad Merhi
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Ahmad Merhi
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type
label
Ahmad Merhi
@ast
Ahmad Merhi
@en
Ahmad Merhi
@es
Ahmad Merhi
@nl
altLabel
Ahmad Merhi
@en
prefLabel
Ahmad Merhi
@ast
Ahmad Merhi
@en
Ahmad Merhi
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Ahmad Merhi
@nl
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P21
P31
P496
0000-0001-7046-2508