about
Protein phosphatase 2A (PP2A) regulates interleukin-4-mediated STAT6 signalingCytotoxicity of CD56(bright) NK cells towards autologous activated CD4+ T cells is mediated through NKG2D, LFA-1 and TRAIL and dampened via CD94/NKG2AIL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.MiR137 is an androgen regulated repressor of an extended network of transcriptional coregulators.cMyc/miR-125b-5p signalling determines sensitivity to bortezomib in preclinical model of cutaneous T-cell lymphomas.Vitamin D up-regulates the vitamin D receptor by protecting it from proteasomal degradation in human CD4+ T cells.Ectopic expression of embryonic stem cell and other developmental genes in cutaneous T-cell lymphomaVitamin D-binding protein controls T cell responses to vitamin D.Midline 1 controls polarization and migration of murine cytotoxic T cells.NKG2D-dependent activation of dendritic epidermal T cells in contact hypersensitivity.Nonmalignant T cells stimulate growth of T-cell lymphoma cells in the presence of bacterial toxins.The effect of short-chain fatty acids on human monocyte-derived dendritic cellsMalignant T cells express lymphotoxin α and drive endothelial activation in cutaneous T cell lymphoma.Analysis of STAT4 expression in cutaneous T-cell lymphoma (CTCL) patients and patient-derived cell linesSOCS5 is expressed in primary B and T lymphoid cells but is dispensable for lymphocyte production and function.Haematopoietic stem cells and their nichesSTAT5-mediated expression of oncogenic miR-155 in cutaneous T-cell lymphoma.Ectopic expression of B-lymphoid kinase in cutaneous T-cell lymphoma.Investigating potential exogenous tumor initiating and promoting factors for Cutaneous T-Cell Lymphomas (CTCL), a rare skin malignancyVitamin D Counteracts Mycobacterium tuberculosis-Induced Cathelicidin Downregulation in Dendritic Cells and Allows Th1 Differentiation and IFNγ SecretionMalignant T cells secrete galectins and induce epidermal hyperproliferation and disorganized stratification in a skin model of cutaneous T-cell lymphoma.Malignant transformation of CD4+ T lymphocytes mediated by oncogenic kinase NPM/ALK recapitulates IL-2-induced cell signaling and gene expression reprogrammingAndrogen dependent mechanisms of pro-angiogenic networks in placental and tumor development.TCR down-regulation boosts T-cell-mediated cytotoxicity and protection against poxvirus infections.Notch1 as a potential therapeutic target in cutaneous T-cell lymphoma.Deficient SOCS3 and SHP-1 expression in psoriatic T cells.Gene variation in IL-7 receptor (IL-7R)α affects IL-7R response in CD4+ T cells in HIV-infected individuals.The immune privilege of the eye: human retinal pigment epithelial cells selectively modulate T-cell activation in vitro.Masking of the CD3 gamma di-leucine-based motif by zeta is required for efficient T-cell receptor expression.TCR comodulation of nonengaged TCR takes place by a protein kinase C and CD3 gamma di-leucine-based motif-dependent mechanism.SHP2 regulates IL-2 induced MAPK activation, but not Stat3 or Stat5 tyrosine phosphorylation, in cutaneous T cell lymphoma cells.Human retinal pigment epithelial cells inhibit proliferation and IL2R expression of activated T cells.The CD3 gamma leucine-based receptor-sorting motif is required for efficient ligand-mediated TCR down-regulation.Endo- and exocytic rate constants for spontaneous and protein kinase C-activated T cell receptor cycling.Ubiquitin-specific protease 2 decreases p53-dependent apoptosis in cutaneous T-cell lymphoma.
P50
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P50
description
onderzoeker
@nl
name
Niels Ødum
@ast
Niels Ødum
@en
Niels Ødum
@es
Niels Ødum
@sl
type
label
Niels Ødum
@ast
Niels Ødum
@en
Niels Ødum
@es
Niels Ødum
@sl
prefLabel
Niels Ødum
@ast
Niels Ødum
@en
Niels Ødum
@es
Niels Ødum
@sl