Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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HDAC1 links early life stress to schizophrenia-like phenotypes Lasting effects of early life stress in mice: interaction of maternal environment and infant genes.

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Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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P2093

Claudia Schmauss

http://www.w3.org/2001/XMLSchema#string

Samantha M Adler

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P2860

HDAC2 negatively regulates memory formation and synaptic plasticity.

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Neurotrophin signal transduction in the nervous system.

Rapid activity-induced transcription of Arc and other IEGs relies on poised RNA polymerase II

Mice lacking dopamine D2 and D3 receptors have spatial working memory deficits

Cortical inhibitory neurons and schizophrenia

Sustained hippocampal chromatin regulation in a mouse model of depression and antidepressant action

Recovery of learning and memory is associated with chromatin remodelling

Too little and too much: hypoactivation and disinhibition of medial prefrontal cortex cause attentional deficits.

Inhibitors of class 1 histone deacetylases reverse contextual memory deficits in a mouse model of Alzheimer's disease.

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1-9

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scholarly article

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10.1016/J.NBD.2016.05.018

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2016-05-31T00:00:00Z

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27260837

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4983517

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Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

about

P2860

P2860

Cognitive deficits triggered by early life stress: The role of histone deacetylase 1.

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