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Extracellular Matrix and Dermal Fibroblast Function in the Healing WoundTopical Collagen-Based Biomaterials for Chronic Wounds: Rationale and Clinical ApplicationProgress toward Treatment and Cure of Epidermolysis Bullosa: Summary of the DEBRA International Research Symposium EB2015Advances in understanding and treating dystrophic epidermolysis bullosaLysyl Hydroxylase 3 Localizes to Epidermal Basement Membrane and Is Reduced in Patients with Recessive Dystrophic Epidermolysis BullosaPolymeric Nanoparticles to Combat Squamous Cell Carcinomas in Patients with Dystrophic Epidermolysis Bullosa.Expression of human skin-specific genes defined by transcriptomics and antibody-based profilingLosartan ameliorates dystrophic epidermolysis bullosa and uncovers new disease mechanismsLentiviral Engineered Fibroblasts Expressing Codon-Optimized COL7A1 Restore Anchoring Fibrils in RDEB.Advancing biomaterials of human origin for tissue engineering.From marrow to matrix: novel gene and cell therapies for epidermolysis bullosa.Transdermal Delivery of Functional Collagen Via Polyvinylpyrrolidone MicroneedlesSuppression of TGFβ and Angiogenesis by Type VII Collagen in Cutaneous SCC.Therapies for inherited skin fragility disorders.Proteomic characterization of platelet gel releasate from adult peripheral and cord blood.Next generation human skin constructs as advanced tools for drug development.miR-29 Regulates Type VII Collagen in Recessive Dystrophic Epidermolysis Bullosa.Analysis of the functional consequences of targeted exon deletion in COL7A1 reveals prospects for dystrophic epidermolysis bullosa therapy.Rescue of the mucocutaneous manifestations by human cord blood derived nonhematopoietic stem cells in a mouse model of recessive dystrophic epidermolysis bullosa.High Local Concentrations of Intradermal MSCs Restore Skin Integrity and Facilitate Wound Healing in Dystrophic Epidermolysis Bullosa.Pseudosyndactyly - an inflammatory and fibrotic wound healing disorder in recessive dystrophic epidermolysis bullosa.Small-molecule therapies for genetic skin fragility.Monozygotic twins discordant for recessive dystrophic epidermolysis bullosa phenotype highlight the role of TGF-β signalling in modifying disease severity.Interaction of Complement Defence Collagens C1q and Mannose-Binding Lectin with BMP-1/Tolloid-like Proteinases.Impaired lymphoid extracellular matrix impedes antibacterial immunity in epidermolysis bullosa.Angiotensin-converting enzyme inhibitor reduces scar formation by inhibiting both canonical and noncanonical TGF-β1 pathways.Combinatorial omics analysis reveals perturbed lysosomal homeostasis in collagen VII-deficient keratinocytes.Extracorporal Shock Waves Activate Migration, Proliferation and Inflammatory Pathways in Fibroblasts and Keratinocytes, and Improve Wound Healing in an Open-Label, Single-Arm Study in Patients with Therapy-Refractory Chronic Leg Ulcers.Genetically corrected iPSCs as cell therapy for recessive dystrophic epidermolysis bullosa.
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Collagen VII plays a dual role in wound healing.
@en
type
label
Collagen VII plays a dual role in wound healing.
@en
prefLabel
Collagen VII plays a dual role in wound healing.
@en
P2093
P2860
P50
P356
P1476
Collagen VII plays a dual role in wound healing.
@en
P2093
Anja Fritsch
Daniela Velati
Johannes S Kern
Venugopal R Mittapalli
P2860
P304
P356
10.1172/JCI68127
P407
P577
2013-07-08T00:00:00Z