Abnormal muscle development in the heldup3 mutant of Drosophila melanogaster is caused by a splicing defect affecting selected troponin I isoforms.
about
Mutations in genes encoding fast-twitch contractile proteins cause distal arthrogryposis syndromesCF2 represses Actin 88F gene expression and maintains filament balance during indirect flight muscle development in Drosophila.Disruption of the protein kinase N gene of drosophila melanogaster results in the recessive delorean allele (pkndln) with a negative impact on wing morphogenesisDrosophila muscle regulation characterized by electron microscopy and three-dimensional reconstruction of thin filament mutants.Functional recovery of troponin I in a Drosophila heldup mutant after a second site mutation.The haplolethal region at the 16F gene cluster of Drosophila melanogaster: structure and functionA direct screen identifies new flight muscle mutants on the Drosophila second chromosome.Suppression of muscle hypercontraction by mutations in the myosin heavy chain gene of Drosophila melanogaster.Flight muscle function in Drosophila requires colocalization of glycolytic enzymes.The RNA-binding protein Arrest (Bruno) regulates alternative splicing to enable myofibril maturation in Drosophila flight muscleA cis-regulatory mutation in troponin-I of Drosophila reveals the importance of proper stoichiometry of structural proteins during muscle assembly.Invertebrate muscles: muscle specific genes and proteins.scully, an essential gene of Drosophila, is homologous to mammalian mitochondrial type II L-3-hydroxyacyl-CoA dehydrogenase/amyloid-beta peptide-binding proteinA tropomyosin-2 mutation suppresses a troponin I myopathy in Drosophila.Specific myosin heavy chain mutations suppress troponin I defects in Drosophila musclesTranscription of Drosophila troponin I gene is regulated by two conserved, functionally identical, synergistic elementsRegulating the contraction of insect flight muscle.Organization of serpin gene-1 from Manduca sexta. Evolution of a family of alternate exons encoding the reactive site loop.Electron microscopy and three-dimensional reconstruction of native thin filaments reveal species-specific differences in regulatory strand densities.In vitro motility of native thin filaments from Drosophila indirect flight muscles reveals that the held-up 2 TnI mutation affects calcium activation.Troponin I is required for myofibrillogenesis and sarcomere formation in Drosophila flight muscle.Tissue expression of four troponin I genes and their molecular interactions with two troponin C isoforms in Caenorhabditis elegans.Roles of the troponin isoforms during indirect flight muscle development in Drosophila.
P2860
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P2860
Abnormal muscle development in the heldup3 mutant of Drosophila melanogaster is caused by a splicing defect affecting selected troponin I isoforms.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Abnormal muscle development in ...... selected troponin I isoforms.
@en
Abnormal muscle development in ...... selected troponin I isoforms.
@nl
type
label
Abnormal muscle development in ...... selected troponin I isoforms.
@en
Abnormal muscle development in ...... selected troponin I isoforms.
@nl
prefLabel
Abnormal muscle development in ...... selected troponin I isoforms.
@en
Abnormal muscle development in ...... selected troponin I isoforms.
@nl
P2093
P2860
P356
P1476
Abnormal muscle development in ...... selected troponin I isoforms.
@en
P2093
P2860
P304
P356
10.1128/MCB.13.3.1433
P407
P577
1993-03-01T00:00:00Z