about
PodocytesEstablishment of protein delivery systems targeting podocytesStructure of the kidney slit diaphragm adapter protein CD2-associated protein as determined with electron microscopy.Small molecule agonists of integrin CD11b/CD18 do not induce global conformational changes and are significantly better than activating antibodies in reducing vascular injury.Soluble Urokinase Receptor and the Kidney Response in Diabetes Mellitus.Role of podocyte B7-1 in diabetic nephropathyEnzymatic disease of the podocyte.Prkdc participates in mitochondrial genome maintenance and prevents Adriamycin-induced nephropathy in mice.Apoptosis, mastocytosis, and diminished adipocytokine gene expression accompany reduced epididymal fat mass in long-standing diet-induced obese miceLeptin deficiency-induced obesity affects the density of mast cells in abdominal fat depots and lymph nodes in miceA Podocyte-Based Automated Screening Assay Identifies Protective Small Molecules.Soluble Urokinase Receptor and Chronic Kidney Disease.Transient receptor potential channel 6 (TRPC6) protects podocytes during complement-mediated glomerular disease.CD11b activation suppresses TLR-dependent inflammation and autoimmunity in systemic lupus erythematosus.Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.Apoptosis and Compensatory Proliferation Signaling Are Coupled by CrkI-Containing Microvesicles.A High-Content Screening Technology for Quantitatively Studying Podocyte Dynamics.A tripartite complex of suPAR, APOL1 risk variants and αvβ3 integrin on podocytes mediates chronic kidney disease.Podocyte GTPases regulate kidney filter dynamics.Mast cells, macrophages, and crown-like structures distinguish subcutaneous from visceral fat in miceCD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survival.Podocytes Exhibit a Specialized Protein Quality Control Employing Derlin-2 in Kidney Disease.High content screening assay based discovery of paullones as novel podocyte protective agents.TRPC5 Does Not Cause or Aggravate Glomerular Disease.Bridges to cross, burn, and mend: cells of renin lineage as podocyte progenitors.Kinetic modeling to optimize pentose fermentation in Zymomonas mobilis.Single nephron proteomes connect morphology and function in proteinuric kidney disease.Modification of kidney barrier function by the urokinase receptor.Purification of TaqI endonuclease from Thermus aquaticusInduction of TRPC6 channel in acquired forms of proteinuric kidney diseasePodocytes: Way to GoMore expression, less function: cleaved dynamin in glomerular kidney diseaseNonimmune cell-derived ICOS ligand functions as a renoprotective αvβ3 integrin-selective antagonist
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P50
description
hulumtues
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onderzoeker
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հետազոտող
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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Mehmet M. Altintas
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P106
P1153
9745693100
P21
P2798
P31
P496
0000-0002-1871-6985