about
Continued stabilization of the nuclear higher-order structure of post-mitotic neurons in vivoGene positional changes relative to the nuclear substructure correlate with the proliferating status of hepatocytes during liver regenerationThe higher-order structure in the cells nucleus as the structural basis of the post-mitotic state.DNA Length Modulates the Affinity of Fragments of Genomic DNA for the Nuclear Matrix In Vitro.The organization of a large transcriptional unit (Fyn) into structural DNA loops is cell-type specific and independent of transcription.Cell-type-specific organization of nuclear DNA into structural looped domains.A global but stable change in HeLa cell morphology induces reorganization of DNA structural loop domains within the cell nucleus.Why Cortical Neurons Cannot Divide, and Why Do They Usually Die in the Attempt?The normal association between newly replicated DNA and the nuclear matrix is abolished in cells infected by herpes simplex virus type 1.Possible cell-free prion replication.DNA moves sequentially towards the nuclear matrix during DNA replication in vivo.The post-mitotic state in neurons correlates with a stable nuclear higher-order structure.A role for the nucleotype in the pathogenesis of primary hepatocellular carcinoma.Loss of DNA loop supercoiling and organization in cells infected by herpes simplex virus type 1.Gene positional changes relative to the nuclear substructure during carbon tetrachloride-induced hepatic fibrosis in rats.Altered chromatin higher-order structure in cells infected by herpes simplex virus type 1.The nuclear higher-order structure defined by the set of topological relationships between DNA and the nuclear matrix is species-specific in hepatocytes.The Set of Structural DNA-Nuclear Matrix Interactions in Neurons Is Cell-Type Specific and Rather Independent of Functional Constraints.Understanding cancer as a formless phenomenon.The interphase mammalian chromosome as a structural system based on tensegrity.Reorganization of the DNA-nuclear matrix interactions in a 210 kb genomic region centered on c-myc after DNA replication in vivo.Cancer development and progression: a non-adaptive process driven by genetic drift.Positional mapping of specific DNA sequences relative to the nuclear substructure by direct polymerase chain reaction on nuclear matrix-bound templatesp53 is a rate-limiting factor in the repair of higher-order DNA structureNatural ageing in the rat liver correlates with progressive stabilisation of DNA-nuclear matrix interactions and withdrawal of genes from the nuclear substructureNeuN/Fox-3 is an intrinsic component of the neuronal nuclear matrixAged and post-mitotic cells share a very stable higher-order structure in the cell nucleus in vivoThe epicenter of chromosomal fragility of Fra14A2, the mouse ortholog of human FRA3B common fragile site, is largely attached to the nuclear matrix in lymphocytes but not in other cell types that do not express such a fragilityLessons we can learn from neurons to make cancer cells quiescent
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description
Mexican biologist
@en
biólogo mexicano
@es
onderzoeker
@nl
name
Armando Aranda-Anzaldo
@ast
Armando Aranda-Anzaldo
@en
Armando Aranda-Anzaldo
@es
Armando Aranda-Anzaldo
@nl
type
label
Armando Aranda-Anzaldo
@ast
Armando Aranda-Anzaldo
@en
Armando Aranda-Anzaldo
@es
Armando Aranda-Anzaldo
@nl
prefLabel
Armando Aranda-Anzaldo
@ast
Armando Aranda-Anzaldo
@en
Armando Aranda-Anzaldo
@es
Armando Aranda-Anzaldo
@nl
P106
P214
P244
P1053
A-6513-2014
P1153
55989779400
P21
P214
P244
P31
P3233
armando-aranda-anzaldo
P3829
P496
0000-0002-5583-5465
P735
P7859
lccn-n87905675