Linear motifs confer functional diversity onto splice variants.
about
Classification of intrinsically disordered regions and proteinsThe new (dis)order in RNA regulationThe contribution of intrinsically disordered regions to protein function, cellular complexity, and human diseaseExon-phase symmetry and intrinsic structural disorder promote modular evolution in the human genome.The "histone mimicry" by pathogens.Evolution at protein ends: major contribution of alternative transcription initiation and termination to the transcriptome and proteome diversity in mammals.Extracting reaction networks from databases-opening Pandora's box.Alternative splicing tends to avoid partial removals of protein-protein interaction sites.Structural disorder provides increased adaptability for vesicle trafficking pathways.The Functional Human C-TerminomeExtent of pre-translational regulation for the control of nucleocytoplasmic protein localization.Plasma proteomics, the Human Proteome Project, and cancer-associated alternative splice variant proteins.Conserved Sequence Preferences Contribute to Substrate Recognition by the Proteasome.Drift and conservation of differential exon usage across tissues in primate species.Computational analysis of translational readthrough proteins in Drosophila and yeast reveals parallels to alternative splicing.Redefining the BH3 Death Domain as a 'Short Linear Motif'.The ribosome-engaged landscape of alternative splicing.Promiscuity as a functional trait: intrinsically disordered regions as central players of interactomes.Complexity of the alternative splicing landscape in plants.How do disordered regions achieve comparable functions to structured domains?Identification of protein features encoded by alternative exons using Exon Ontology.SLiMSearch: a framework for proteome-wide discovery and annotation of functional modules in intrinsically disordered regions.Interpreting functional effects of coding variants: challenges in proteome-scale prediction, annotation and assessment.A highly conserved program of neuronal microexons is misregulated in autistic brains.The switches.ELM resource: a compendium of conditional regulatory interaction interfaces.Fuzziness enables context dependence of protein interactions.
P2860
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P2860
Linear motifs confer functional diversity onto splice variants.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Linear motifs confer functional diversity onto splice variants.
@en
Linear motifs confer functional diversity onto splice variants.
@nl
type
label
Linear motifs confer functional diversity onto splice variants.
@en
Linear motifs confer functional diversity onto splice variants.
@nl
prefLabel
Linear motifs confer functional diversity onto splice variants.
@en
Linear motifs confer functional diversity onto splice variants.
@nl
P2860
P50
P356
P1476
Linear motifs confer functional diversity onto splice variants.
@en
P2860
P304
P356
10.1093/NAR/GKS442
P577
2012-05-25T00:00:00Z