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Differential effects of 670 and 830 nm red near infrared irradiation therapy: a comparative study of optic nerve injury, retinal degeneration, traumatic brain and spinal cord injury.Strategies to limit dysmyelination during secondary degeneration following neurotraumaEarly proliferation does not prevent the loss of oligodendrocyte progenitor cells during the chronic phase of secondary degeneration in a CNS white matter tract.Paranode Abnormalities and Oxidative Stress in Optic Nerve Vulnerable to Secondary Degeneration: Modulation by 670 nm Light TreatmentIron oxide-induced thermal effects on solid-state upconversion emissions in NaYF4:Yb,Er nanocrystals.Changes to mitochondrial ultrastructure in optic nerve vulnerable to secondary degeneration in vivo are limited by irradiation at 670 nmThe influence of NaYF₄:Yb,Er size/phase on the multimodality of co-encapsulated magnetic photon-upconverting polymeric nanoparticles.Method for the assessment of effects of a range of wavelengths and intensities of red/near-infrared light therapy on oxidative stress in vitroRedefining the role of metallothionein within the injured brain: extracellular metallothioneins play an important role in the astrocyte-neuron response to injury.Elevation of oxidative stress indicators in a pilot study of plasma following traumatic brain injury.A new perspective on delivery of red-near-infrared light therapy for disorders of the brain.An Unexpected Transient Breakdown of the Blood Brain Barrier Triggers Passage of Large Intravenously Administered Nanoparticles.Comparative assessment of phototherapy protocols for reduction of oxidative stress in partially transected spinal cord slices undergoing secondary degeneration.Delayed treatment of secondary degeneration following acute optic nerve transection using a combination of ion channel inhibitors.Neuroprotective peptides fused to arginine-rich cell penetrating peptides: Neuroprotective mechanism likely mediated by peptide endocytic properties.Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma.Characterization of polymeric nanoparticles for treatment of partial injury to the central nervous system.Repeated mild traumatic brain injury in female rats increases lipid peroxidation in neurons.Specific combinations of ion channel inhibitors reduce excessive Ca(2+) influx as a consequence of oxidative stress and increase neuronal and glial cell viability in vitro.Mineralocorticoids restore quiescent morphology and reduce VEGF receptor expression in inflamed choroidal endothelial cells in vitro.Angiostatin formation involves disulfide bond reduction and proteolysis in kringle 5 of plasmin.Prolonged glutamate excitotoxicity increases GluR1 immunoreactivity but decreases mRNA of GluR1 and associated regulatory proteins in dissociated rat retinae in vitro.The complex contribution of the astrocyte scar.Near infrared light reduces oxidative stress and preserves function in CNS tissue vulnerable to secondary degeneration following partial transection of the optic nerve.Early events of secondary degeneration after partial optic nerve transection: an immunohistochemical study.Spinal dorsal horn cell receptive field size is increased in adult rats following neonatal hindpaw skin injury.Antioxidant phospholipid calix[4]arene mimics as micellular delivery systems.Secondary degeneration of the optic nerve following partial transection: the benefits of lomerizine.Limiting oxidative stress following neurotrauma with a combination of ion channel inhibitors.Neurite responses to ephrin-A5 modulated by BDNF: evidence for TrkB-EphA interactions.Structural requirements for anti-oxidant activity of calix[n]arenes and their associated anti-bacterial activity.Reactive species and oxidative stress in optic nerve vulnerable to secondary degeneration.The Protein Corona of PEGylated PGMA-Based Nanoparticles is Preferentially Enriched with Specific Serum Proteins of Varied Biological Function.Resistance wheel exercise from mid-life has minimal effect on sciatic nerves from old mice in which sarcopenia was prevented.Three Ca2+ channel inhibitors in combination limit chronic secondary degeneration following neurotrauma.Metallothionein-IIA promotes neurite growth via the megalin receptor.Abnormal strategies during visual discrimination reversal learning in ephrin-A2(-/-) mice.Novel combinations of ion channel inhibitors for treatment of neurotrauma.In vivo imaging and biodistribution of multimodal polymeric nanoparticles delivered to the optic nerve.The effects of concentration-dependent morphology of self-assembling RADA16 nanoscaffolds on mixed retinal cultures.
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Melinda Fitzgerald
@ast
Melinda Fitzgerald
@en
Melinda Fitzgerald
@es
Melinda Fitzgerald
@nl
Melinda Fitzgerald
@sl
type
label
Melinda Fitzgerald
@ast
Melinda Fitzgerald
@en
Melinda Fitzgerald
@es
Melinda Fitzgerald
@nl
Melinda Fitzgerald
@sl
prefLabel
Melinda Fitzgerald
@ast
Melinda Fitzgerald
@en
Melinda Fitzgerald
@es
Melinda Fitzgerald
@nl
Melinda Fitzgerald
@sl
P1053
C-4235-2011
P106
P1153
7402773604
P21
P31
P3829
P496
0000-0002-4823-8179