Opposing effects of PSD-93 and PSD-95 on long-term potentiation and spike timing-dependent plasticity.
about
Leucine-rich repeat transmembrane proteins are essential for maintenance of long-term potentiationThe molecular evolution of the vertebrate behavioural repertoireHippocampus-based contextual memory alters the morphological characteristics of astrocytes in the dentate gyrus.Molecular and genetic determinants of the NMDA receptor for superior learning and memory functionsTargeted tandem affinity purification of PSD-95 recovers core postsynaptic complexes and schizophrenia susceptibility proteinsDlg5 regulates dendritic spine formation and synaptogenesis by controlling subcellular N-cadherin localization.Diversification of behavior and postsynaptic properties by netrin-G presynaptic adhesion family proteins.Impaired synaptic clustering of postsynaptic density proteins and altered signal transmission in hippocampal neurons, and disrupted learning behavior in PDZ1 and PDZ2 ligand binding-deficient PSD-95 knockin miceEvolution of GluN2A/B cytoplasmic domains diversified vertebrate synaptic plasticity and behaviorHistory of the Concept of Disconnectivity in Schizophrenia.BAI1 regulates spatial learning and synaptic plasticity in the hippocampusExperimental and computational aspects of signaling mechanisms of spike-timing-dependent plasticity.Narrowing the boundaries of the genetic architecture of schizophrenia.Autism-like behaviours and enhanced memory formation and synaptic plasticity in Lrfn2/SALM1-deficient miceElectromagnetic field effect or simply stress? Effects of UMTS exposure on hippocampal longterm plasticity in the context of procedure related hormone release.Synaptic scaffold evolution generated components of vertebrate cognitive complexity.Beta-adrenergic receptor activation rescues theta frequency stimulation-induced LTP deficits in mice expressing C-terminally truncated NMDA receptor GluN2A subunits.Hippocampal dysregulation of synaptic plasticity-associated proteins with age-related cognitive decline.Knockdown of mental disorder susceptibility genes disrupts neuronal network physiology in vitroIQGAP1 regulates NR2A signaling, spine density, and cognitive processes.PSD-95-like membrane associated guanylate kinases (PSD-MAGUKs) and synaptic plasticity.PSD-95 is post-transcriptionally repressed during early neural development by PTBP1 and PTBP2.Misaligned feeding impairs memories.Coordinated activation of distinct Ca(2+) sources and metabotropic glutamate receptors encodes Hebbian synaptic plasticity.Eye opening and PSD95 are required for long-term potentiation in developing superior colliculusTherapeutic testosterone administration preserves excitatory synaptic transmission in the hippocampus during autoimmune demyelinating diseaseTRPA1 channels are regulators of astrocyte basal calcium levels and long-term potentiation via constitutive D-serine release.Synaptic state-dependent functional interplay between postsynaptic density-95 and synapse-associated protein 102.Modulation of behavior by scaffolding proteins of the post-synaptic density.Differential roles of postsynaptic density-93 isoforms in regulating synaptic transmission.Retinoic acid modulates intrahippocampal levels of corticosterone in middle-aged mice: consequences on hippocampal plasticity and contextual memory.Differential requirement for NMDAR activity in SAP97β-mediated regulation of the number and strength of glutamatergic AMPAR-containing synapses.NMDA receptors are selectively partitioned into complexes and supercomplexes during synapse maturation.TNiK is required for postsynaptic and nuclear signaling pathways and cognitive functionThe activity requirements for spike timing-dependent plasticity in the hippocampus.The postsynaptic organization of synapses.Dopamine regulates intrinsic excitability thereby gating successful induction of spike timing-dependent plasticity in CA1 of the hippocampus.Synaptic localization of neurotransmitter receptors: comparing mechanisms for AMPA and GABAA receptors.Novel promoters and coding first exons in DLG2 linked to developmental disorders and intellectual disability.Sharp-Wave Ripples Orchestrate the Induction of Synaptic Plasticity during Reactivation of Place Cell Firing Patterns in the Hippocampus.
P2860
Q26269850-3415D741-CD59-4484-962B-9E3265C2BA4FQ26776358-D7414667-AEAA-4129-9878-2CF6D0157E70Q27300718-F118F593-508C-4B09-A406-970934998FE4Q27332137-D033303F-CF4E-4243-927A-912B6179F8CEQ28585023-1040DEFB-8BB6-4F08-9F04-30D6C997A83CQ29871120-997D5B7C-9194-4AA5-810C-10A6F82F34F6Q30394198-F60671F8-4A4E-44FD-820F-3D2CD8C4C3DAQ30458150-DFE3E821-6E32-4464-93E4-C76989A2776EQ30575656-802E05BA-884F-478F-A2D3-2AE1DCA0F3B6Q31054567-C6417F2F-7F07-4287-B54B-73981137004CQ33112266-3F91BCBF-8E18-4CC3-BD0F-2C60843C505AQ33565311-49B62327-F4D5-4D3F-B2BF-FA7CBD6466EDQ33566158-45705085-37E9-496F-AD0E-AB3ADAE14DEBQ33804802-2BC075D2-2689-4E71-BF09-BD835C37EA77Q33900875-8F69AE70-33D1-4EE5-B64C-8902209FF934Q34038754-460F50E4-9C94-4686-A2F2-4EA097835122Q34546616-B830DA06-D718-4071-98C4-E245B0436232Q34988032-A4EBCC30-4821-44B0-86B2-560D0BE4FB1DQ35017276-A1ED850C-25AC-432E-A5CC-6F07BD6A8C05Q35062626-8F000052-70CF-4543-B28A-A9596D2E7845Q35110479-8E95CCC7-BE9A-44C5-9FD7-B909350B4C35Q35783819-69DC72AE-FDDA-40FD-B02B-AEB909DAE748Q36510606-C919664D-1510-4891-907B-DE8A3314CDCFQ36527476-39714C73-EBB2-4B94-96EF-2B730C5F067BQ36535208-D36AB929-7352-4737-84E3-69DC2ED0BB4EQ36608070-B8DC82DF-C8A2-4268-AD24-A3B396693A82Q36928480-7920F744-C089-44D9-9440-2DABF95B1F7DQ37093883-88C7A0A7-3283-4945-8A36-6A57D22333AEQ37161173-F770F6B3-2E85-49DB-AAE9-A6998040B435Q37196313-B7D5F507-32CA-4035-9AB4-8E7B4D063BA4Q37565705-38AA15FF-BA12-438B-8C7A-285030FB0C82Q37578416-2FC7BCA7-6047-4018-85BB-0A4E2A81308CQ37580996-C41DA00C-C110-4B35-8C6D-EB4AD19B4727Q37690413-0951ACE3-9FAE-4A65-AD9D-0BE4ACEE68C5Q37855558-B16EFAD9-00C3-4719-8FCF-D6864E0FDC7CQ37951923-6CB9E2EF-90E9-4C45-B745-8D16512DCD4AQ38090956-4823E72A-BC54-400C-9D51-7286ED3F0197Q38295906-A20F5D32-36BC-4010-AD82-0042FAD16DB5Q38671614-3C9301B7-E9A1-4F8B-9EE3-DA5A517D59E0Q38899990-564A7540-66DE-4D90-9CE6-5701FE4AF5D3
P2860
Opposing effects of PSD-93 and PSD-95 on long-term potentiation and spike timing-dependent plasticity.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@en
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@nl
type
label
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@en
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@nl
prefLabel
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@en
Opposing effects of PSD-93 and ...... e timing-dependent plasticity.
@nl
P2860
P1476
Opposing effects of PSD-93 and ...... ke timing-dependent plasticity
@en
P2093
Holly J Carlisle
Thomas J O'Dell
P2860
P304
P356
10.1113/JPHYSIOL.2008.163469
P407
P577
2008-10-20T00:00:00Z