about
Crigiau oddi cartrefCore signaling pathways in human pancreatic cancers revealed by global genomic analysesDistant metastasis occurs late during the genetic evolution of pancreatic cancerIntegrated genomic analyses identify ARID1A and ARID1B alterations in the childhood cancer neuroblastomaAn integrated genomic analysis of human glioblastoma multiformeIDH1 and IDH2 mutations in gliomasIntegrated analysis of homozygous deletions, focal amplifications, and sequence alterations in breast and colorectal cancersGenetic mutations associated with cigarette smoking in pancreatic cancerExomic sequencing identifies PALB2 as a pancreatic cancer susceptibility geneFunctional characterization of two human MutY homolog (hMYH) missense mutations (R227W and V232F) that lie within the putative hMSH6 binding domain and are associated with hMYH polyposisSMAD4 gene mutations are associated with poor prognosis in pancreatic cancer.Personalizing cancer treatment in the age of global genomic analyses: PALB2 gene mutations and the response to DNA damaging agents in pancreatic cancer.Expression of p16 and retinoblastoma determines response to CDK4/6 inhibition in ovarian cancer.The consensus coding sequences of human breast and colorectal cancersThe genomic landscapes of human breast and colorectal cancersBiallelic germline mutations in MYH predispose to multiple colorectal adenoma and somatic G:C-->T:A mutationsFrequent mutations of chromatin remodeling gene ARID1A in ovarian clear cell carcinomaThe genetic landscape of the childhood cancer medulloblastomaSomatic mutations in the chromatin remodeling gene ARID1A occur in several tumor types.Genome-wide linkage scan for colorectal cancer susceptibility genes supports linkage to chromosome 3q.Exome sequencing identifies somatic gain-of-function PPM1D mutations in brainstem gliomas.Clinical implications of genomic alterations in the tumour and circulation of pancreatic cancer patientsIntegrated next-generation sequencing and avatar mouse models for personalized cancer treatment.Genomic analyses of gynaecologic carcinosarcomas reveal frequent mutations in chromatin remodelling genesFamilial and sporadic pancreatic cancer share the same molecular pathogenesis.Radiofrequency ablation for treatment of symptomatic uterine fibroids.Personalized genomic analyses for cancer mutation discovery and interpretation.A multidimensional analysis of genes mutated in breast and colorectal cancers.CoGAPS matrix factorization algorithm identifies transcriptional changes in AP-2alpha target genes in feedback from therapeutic inhibition of the EGFR network.Clinical significance of the genetic landscape of pancreatic cancer and implications for identification of potential long-term survivors.Low-grade serous carcinomas of the ovary contain very few point mutationsComparative lesion sequencing provides insights into tumor evolutionFrequent activating mutations of PIK3CA in ovarian clear cell carcinoma.Therapeutic potential of the poly(ADP-ribose) polymerase inhibitor rucaparib for the treatment of sporadic human ovarian cancer.Genomic and Immunological Tumor Profiling Identifies Targetable Pathways and Extensive CD8+/PDL1+ Immune Infiltration in Inflammatory Breast Cancer Tumors.Recurrent KRAS codon 146 mutations in human colorectal cancer.Cancer-Associated Mutations in Endometriosis without Cancer.The APC Variant p.Glu1317Gln predisposes to colorectal adenomas by a novel mechanism of relaxing the target for tumorigenic somatic APC mutations.Mutations in linker histone genes HIST1H1 B, C, D, and E; OCT2 (POU2F2); IRF8; and ARID1A underlying the pathogenesis of follicular lymphoma.Crigiau oddi cartref
P50
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P50
name
Siân Jones
@ast
Siân Jones
@en
Siân Jones
@nl
type
label
Siân Jones
@ast
Siân Jones
@en
Siân Jones
@nl
prefLabel
Siân Jones
@ast
Siân Jones
@en
Siân Jones
@nl