about
COMMD1 forms oligomeric complexes targeted to the endocytic membranes via specific interactions with phosphatidylinositol 4,5-bisphosphateCellular multitasking: the dual role of human Cu-ATPases in cofactor delivery and intracellular copper balanceElevated copper remodels hepatic RNA processing machinery in the mouse model of Wilson's diseaseConsequences of copper accumulation in the livers of the Atp7b-/- (Wilson disease gene) knockout miceAnalysis of transgenic Indian mustard plants for phytoremediation of metal-contaminated mine tailings.Wilson disease at a single cell level: intracellular copper trafficking activates compartment-specific responses in hepatocytes.Systems biology approach to Wilson's diseaseNutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of nonalcoholic fatty liver diseaseA systems approach implicates nuclear receptor targeting in the Atp7b(-/-) mouse model of Wilson's disease.Copper homeostasis.Host Cell Copper Transporters CTR1 and ATP7A are important for Influenza A virus replication.Transcriptional activation of glutathione pathways and role of glucose homeostasis during copper imbalance.The role of insufficient copper in lipid synthesis and fatty-liver disease.High copper selectively alters lipid metabolism and cell cycle machinery in the mouse model of Wilson disease.The Arabidopsis thaliana CUTA gene encodes an evolutionarily conserved copper binding chloroplast protein.Altered zinc balance in the Atp7b mouse reveals a mechanism of copper toxicity in Wilson diseaseCopper-dependent amino oxidase 3 governs selection of metabolic fuels in adipocytesHigher plants possess two different types of ATX1-like copper chaperonesAtCCS is a functional homolog of the yeast copper chaperone Ccs1/Lys7COMMD1 and PtdIns(4,5)P2 interaction maintain ATP7B copper transporter trafficking fidelity in HepG2 cellsCopper Deficiency in Liver Diseases: A Case Series and Pathophysiological ConsiderationsChronic copper treatment prevents the liver critical balance transcription response induced by acetaminophen
P50
Q24336648-559B1458-8536-4B3A-B2B4-8AE713ABA714Q24642573-2F54E991-A7D6-4C24-9F67-42779CCFFEC3Q28386083-16BC764F-04F5-47DB-A5FD-FB408A64DA99Q28594419-E58F5B23-17B9-4739-92EE-471F9300DF1AQ30052454-F4EA432E-A686-41A1-A353-3EADC3EBCBE3Q34155396-7809B716-0827-467B-A176-94453CDE950EQ35020396-B2C037C3-60A1-43D6-97B9-0123791A33F2Q36083434-537087FB-1B9F-4E15-9CFF-6D0B6D404054Q36964471-A73F3237-B460-4405-9CBE-FD1FDC6AAEBFQ37464252-29781F72-66F7-4EDE-960F-03956C92834FQ37602529-D2321174-8A47-4FCA-8ADC-8D9F3125A310Q38907434-73558B9C-CC59-4000-818A-365BF517DAF6Q38921695-3DFDEA94-2464-4835-AC4E-373A44CDFE13Q53579211-2BE7BD19-6069-4A68-A3DD-0B8A74CC7A67Q54523762-9DEC0CC9-7814-47CB-A158-8612546234DDQ57048329-AAB9154C-0A8F-4E6B-A82C-333B7E2C01C8Q58756610-BC18B265-F4D0-4635-909B-A64219F352FBQ79558312-F7B811A8-5EB0-4BE0-8487-918C1546B1CAQ81679880-2D224E74-737F-409D-9261-5B7638C349A9Q90083239-EE2A4E95-204F-47EE-BB21-A3D16326794AQ92464107-8ECF66AE-8B54-4D9C-AF7D-4864CF48D2B0Q92609358-68198D30-8D0F-403C-A8BA-C27A06F70132
P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Jason Burkhead
@ast
Jason Burkhead
@en
Jason Burkhead
@es
Jason Burkhead
@nl
Jason Burkhead
@sl
type
label
Jason Burkhead
@ast
Jason Burkhead
@en
Jason Burkhead
@es
Jason Burkhead
@nl
Jason Burkhead
@sl
prefLabel
Jason Burkhead
@ast
Jason Burkhead
@en
Jason Burkhead
@es
Jason Burkhead
@nl
Jason Burkhead
@sl
P1053
C-9612-2013
P106
P21
P31
P3829
P496
0000-0001-5457-311X