Stat6 and Jak1 are common elements in platelet-derived growth factor and interleukin-4 signal transduction pathways in NIH 3T3 fibroblasts.
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Consequences of Stat6 deletion on Sis/PDGF- and IL-4-induced proliferation and transcriptional activation in murine fibroblastsGrowth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasisTransplacental passage of interleukins 4 and 13?Proliferation of adult T cell leukemia/lymphoma cells is associated with the constitutive activation of JAK/STAT proteinsStat6 Promotes Intestinal Tumorigenesis in a Mouse Model of Adenomatous Polyposis by Expansion of MDSCs and Inhibition of Cytotoxic CD8 Response.Insulin-like growth factor I synergizes with interleukin 4 for hematopoietic cell proliferation independent of insulin receptor substrate expression.Regulation of interleukin 4-mediated signaling by naturally occurring dominant negative and attenuated forms of human Stat6.Nuclear Localization and Cleavage of STAT6 Is Induced by Kaposi's Sarcoma-Associated Herpesvirus for Viral Latency.Essential role of signal transducer and activator of transcription (Stat)5a but not Stat5b for Flt3-dependent signaling.Signal transducer and activator of transcription factor 6 (Stat6)-deficient mice are protected from antigen-induced airway hyperresponsiveness and mucus production.Receptor-associated constitutive protein tyrosine phosphatase activity controls the kinase function of JAK1.Dissection of the cytoplasmic domains of cytokine receptors involved in STAT and Ras dependent proliferation.Mitogenic synergy through multilevel convergence of hepatocyte growth factor and interleukin-4 signaling pathways.Multiple growth factor induction of a murine early response gene that complements a lethal defect in yeast ribosome biogenesis.Jak1 expression is required for mediating interleukin-4-induced tyrosine phosphorylation of insulin receptor substrate and Stat6 signaling molecules.Regulation of gene expression, growth, and cell survival by IL-4: contribution of multiple signaling pathways.Platelet-derived growth factor (PDGF)-induced activation of signal transducer and activator of transcription (Stat) 5 is mediated by PDGF beta-receptor and is not dependent on c-src, fyn, jak1 or jak2 kinases.Persea americana Mill. Seed: Fractionation, Characterization, and Effects on Human Keratinocytes and Fibroblasts.Activation of Stat3 preassembled with platelet-derived growth factor beta receptors requires Src kinase activity.STAT activation by the PDGF receptor requires juxtamembrane phosphorylation sites but not Src tyrosine kinase activation.Stat3 plays an important role in oncogenic Ros- and insulin-like growth factor I receptor-induced anchorage-independent growth.Novel mechanisms of Collagenase Santyl Ointment (CSO) in wound macrophage polarization and resolution of wound inflammation.JAK-STAT signaling and the atrial fibrillation promoting fibrotic substrate.
P2860
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P2860
Stat6 and Jak1 are common elements in platelet-derived growth factor and interleukin-4 signal transduction pathways in NIH 3T3 fibroblasts.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@en
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@nl
type
label
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@en
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@nl
prefLabel
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@en
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@nl
P2093
P2860
P356
P1476
Stat6 and Jak1 are common elem ...... thways in NIH 3T3 fibroblasts.
@en
P2093
P2860
P304
22175-22182
P356
10.1074/JBC.271.36.22175
P407
P577
1996-09-01T00:00:00Z