Vertebrate CASTOR is required for differentiation of cardiac precursor cells at the ventral midline.
about
A conserved regulatory logic controls temporal identity in mouse neural progenitorsProteomic profiling of cardiac tissue by isolation of nuclei tagged in specific cell types (INTACT)Congenital heart disease protein 5 associates with CASZ1 to maintain myocardial tissue integrity.The CASZ1/Egfl7 transcriptional pathway is required for RhoA expression in vascular endothelial cells.CASZ1b, the short isoform of CASZ1 gene, coexpresses with CASZ1a during neurogenesis and suppresses neuroblastoma cell growthEssential role of the zinc finger transcription factor Casz1 for mammalian cardiac morphogenesis and developmentRNA-seq in the tetraploid Xenopus laevis enables genome-wide insight in a classic developmental biology model organismXenopus: An emerging model for studying congenital heart disease.CASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression.Casz1 is required for cardiomyocyte G1-to-S phase progression during mammalian cardiac developmentCharacterization of critical domains within the tumor suppressor CASZ1 required for transcriptional regulation and growth suppressionZinc finger transcription factor CASZ1 interacts with histones, DNA repair proteins and recruits NuRD complex to regulate gene transcriptionMorpholino injection in Xenopus.CASZ1 promotes vascular assembly and morphogenesis through the direct regulation of an EGFL7/RhoA-mediated pathway.Castor is required for Hedgehog-dependent cell-fate specification and follicle stem cell maintenance in Drosophila oogenesis.Tcf21 regulates the specification and maturation of proepicardial cells.CASZ1 is a novel promoter of metastasis in ovarian cancer.CASZ1 inhibits cell cycle progression in neuroblastoma by restoring pRb activityMyocardial lineage development.Cellular and molecular mechanisms underlying blood vessel lumen formation.Mechanisms of temporal identity regulation in mouse retinal progenitor cells.Formation of a TBX20-CASZ1 protein complex is protective against dilated cardiomyopathy and critical for cardiac homeostasis.Transcriptional regulation of blood vessel formation: the role of the CASZ1/Egfl7/RhoA pathway.Differential regulation of CASZ1 protein expression during cardiac and skeletal muscle development.Anacardic acid and thyroid hormone enhance cardiomyocytes production from undifferentiated mouse ES cells along functionally distinct pathways.Zinc finger transcription factor Casz1 expression is regulated by homeodomain transcription factor Prrxl1 in embryonic spinal dorsal horn late-born excitatory interneurons.A Matter of the Heart: The African Clawed Frog Xenopus as a Model for Studying Vertebrate Cardiogenesis and Congenital Heart Defects.Identification of Casz1 as a Regulatory Protein Controlling T Helper Cell Differentiation, Inflammation, and Immunity.Downregulation of castor zinc finger 1 predicts poor prognosis and facilitates hepatocellular carcinoma progression via MAPK/ERK signaling.Loss of gene function as a consequence of human papillomavirus DNA integration.Casz1 controls higher-order nuclear organization in rod photoreceptors
P2860
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P2860
Vertebrate CASTOR is required for differentiation of cardiac precursor cells at the ventral midline.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Vertebrate CASTOR is required ...... cells at the ventral midline.
@en
Vertebrate CASTOR is required ...... cells at the ventral midline.
@nl
type
label
Vertebrate CASTOR is required ...... cells at the ventral midline.
@en
Vertebrate CASTOR is required ...... cells at the ventral midline.
@nl
prefLabel
Vertebrate CASTOR is required ...... cells at the ventral midline.
@en
Vertebrate CASTOR is required ...... cells at the ventral midline.
@nl
P2860
P1433
P1476
Vertebrate CASTOR is required ...... cells at the ventral midline.
@en
P2093
Frank L Conlon
Kathleen S Christine
P2860
P304
P356
10.1016/J.DEVCEL.2008.01.009
P407
P577
2008-04-01T00:00:00Z