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Functional programming of the autonomic nervous system by early life immune exposure: implications for anxiety.mTORC1 inhibition in the nucleus accumbens 'protects' against the expression of drug seeking and 'relapse' and is associated with reductions in GluA1 AMPAR and CAMKIIα levels.Tyrosine hydroxylase phosphorylation in catecholaminergic brain regions: a marker of activation following acute hypotension and glucoprivation.Early life peripheral lipopolysaccharide challenge reprograms catecholaminergic neurons.Chronic stress exposure following photothrombotic stroke is associated with increased levels of Amyloid beta accumulation and altered oligomerisation at sites of thalamic secondary neurodegeneration in mice.Neurobiological consequences of acute footshock stress: effects on tyrosine hydroxylase phosphorylation and activation in the rat brain and adrenal medulla.Peripheral Lipopolysaccharide Challenge Induces Long-Term Changes in Tyrosine Hydroxylase Regulation in the Adrenal Medulla.Signal transduction pathways and tyrosine hydroxylase regulation in the adrenal medulla following glucoprivation: an in vivo analysis.Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration.Neonatal overfeeding increases capacity for catecholamine biosynthesis from the adrenal gland acutely and long-term in the male rat.Rapamycin reduces motivated responding for cocaine and alters GluA1 expression in the ventral but not dorsal striatum.Impaired microglia process dynamics post-stroke are specific to sites of secondary neurodegeneration.Early life stress and post-weaning high fat diet alter tyrosine hydroxylase regulation and AT1 receptor expression in the adrenal gland in a sex dependent manner.Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend or foe?Chronic stress induced disruption of the peri-infarct neurovascular unit following experimentally induced photothrombotic stroke.Oral administration of corticosterone at stress-like levels drives microglial but not vascular disturbances post-stroke.Authors' response re: "Reconsidering the role of glial cells in chronic stress-induced dopaminergic neurons loss within the substantia nigra? Friend of foe?" by Ong et al. Brain Behavior and Immunity, 2016.Increased microglial activation in the rat brain following neonatal exposure to a bacterial mimetic.Tyrosine hydroxylase regulation in adult rat striatum following short-term neonatal exposure to manganese.The effects of footshock and immobilization stress on tyrosine hydroxylase phosphorylation in the rat locus coeruleus and adrenal gland.The sustained phase of tyrosine hydroxylase activation in vivo.Growth Hormone Deficiency Is Frequent After Recent StrokeThe Effect of Social Defeat on Tyrosine Hydroxylase Phosphorylation in the Rat Brain and Adrenal GlandSpatiotemporal analysis of impaired microglia process movement at sites of secondary neurodegeneration post-strokeVisual discrimination impairment after experimental stroke is associated with disturbances in the polarization of the astrocytic aquaporin-4 and increased accumulation of neurotoxic proteinsGrowth Hormone Promotes Motor Function after Experimental Stroke and Enhances Recovery-Promoting Mechanisms within the Peri-Infarct Area
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Lin Kooi Ong
@ast
Lin Kooi Ong
@en
Lin Kooi Ong
@es
Lin Kooi Ong
@nl
Lin Kooi Ong
@sl
type
label
Lin Kooi Ong
@ast
Lin Kooi Ong
@en
Lin Kooi Ong
@es
Lin Kooi Ong
@nl
Lin Kooi Ong
@sl
prefLabel
Lin Kooi Ong
@ast
Lin Kooi Ong
@en
Lin Kooi Ong
@es
Lin Kooi Ong
@nl
Lin Kooi Ong
@sl
P1053
H-2797-2012
P106
P1153
56033871000
P21
P31
P3829
P496
0000-0001-8664-0540
P569
2000-01-01T00:00:00Z