Insight into the structural basis of pro- and antiapoptotic p53 modulation by ASPP proteins.
about
Combinatorial control of gene expressionWrecked regulation of intrinsically disordered proteins in diseases: pathogenicity of deregulated regulatorsStructural Basis for ASPP2 Recognition by the Tumor Suppressor p73The ASPP interaction network: electrostatic differentiation between pro- and anti-apoptotic proteinsHighly homologous proteins exert opposite biological activities by using different interaction interfacesLysine120 interactions with p53 response elements can allosterically direct p53 organization.iASPP is over-expressed in human non-small cell lung cancer and regulates the proliferation of lung cancer cells through a p53 associated pathway.The RNA binding protein HuR does not interact directly with HIV-1 reverse transcriptase and does not affect reverse transcription in vitro.Proteomic analysis of pathways involved in estrogen-induced growth and apoptosis of breast cancer cells.In vitro effect of iASPP on cell growth of oral tongue squamous cell carcinoma.The Cullin-RING E3 ubiquitin ligase CRL4-DCAF1 complex dimerizes via a short helical region in DCAF1.Inhibitor of apoptosis-stimulating protein of p53 (iASPP) is required for neuronal survival after axonal injury.Analysis of an ankyrin-like region in Epstein Barr Virus encoded (EBV) BZLF-1 (ZEBRA) protein: implications for interactions with NF-κB and p53.Gene-specific transcription activation via long-range allosteric shape-shifting.The selective BH4-domain biology of Bcl-2-family members: IP3Rs and beyond.Inhibitory member of the apoptosis-stimulating protein of p53 is overexpressed in bladder cancer and correlated to its progression.An indirect role for ASPP1 in limiting p53-dependent p21 expression and cellular senescence.Cytoplasmic ASPP1 inhibits apoptosis through the control of YAP.Multi-scale ensemble modeling of modular proteins with intrinsically disordered linker regions: application to p53.How do transcription factors select specific binding sites in the genome?The roles of intrinsic disorder-based liquid-liquid phase transitions in the "Dr. Jekyll-Mr. Hyde" behavior of proteins involved in amyotrophic lateral sclerosis and frontotemporal lobar degeneration.Epigenetic silencing of ASPP1 confers 5-FU resistance in clear cell renal cell carcinoma by preventing p53 activation.
P2860
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P2860
Insight into the structural basis of pro- and antiapoptotic p53 modulation by ASPP proteins.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@en
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@nl
type
label
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@en
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@nl
prefLabel
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@en
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@nl
P2093
P2860
P356
P1476
Insight into the structural ba ...... 3 modulation by ASPP proteins.
@en
P2093
Chang-Hyeock Byeon
In-Ja L Byeon
Jinwoo Ahn
P2860
P304
13812-13822
P356
10.1074/JBC.M808821200
P407
P577
2009-02-26T00:00:00Z