about
Bone histomorphometry revisitedChronic arthritis leads to disturbances in the bone collagen network.At the moment of occurrence of a fragility hip fracture, men have higher mechanical properties values in comparison with women.Improving the DNA specificity and applicability of base editing through protein engineering and protein delivery.Upregulation of inflammatory genes and downregulation of sclerostin gene expression are key elements in the early phase of fragility fracture healing.Osteoblasts and bone formation.Contribution for new genetic markers of rheumatoid arthritis activity and severity: sequencing of the tumor necrosis factor-alpha gene promoterEffect of Tumor Necrosis Factor Inhibitor Therapy on Osteoclasts Precursors in Ankylosing SpondylitisOsteoimmunology--the hidden immune regulation of bone.Biomechanical effects of inflammatory diseases on bone-rheumatoid arthritis as a paradigm.Ankylosing Spondylitis Patients Have Impaired Osteoclast Gene Expression in Circulating Osteoclast Precursors.Methotrexate and low-dose prednisolone downregulate osteoclast function by decreasing receptor activator of nuclear factor-κβ expression in monocytes from patients with early rheumatoid arthritis.Effect of Tumor Necrosis Factor Inhibitor Therapy on Osteoclasts Precursors in Rheumatoid Arthritis.Alterations on peripheral blood B-cell subpopulations in very early arthritis patients.An analysis of joint replacement in patients with systemic lupus erythematosus.Tumor necrosis factor-alpha -308 genotypes influence inflammatory activity and TNF-alpha serum concentrations in children with juvenile idiopathic arthritis.Protective effect of an ERAP1 haplotype in ankylosing spondylitis: investigating non-MHC genes in HLA-B27-positive individuals.Apolipoprotein E and undercaboxylated osteocalcin are associated with bone fragility but not with bone mineral density in osteoarthritis patients.Delayed neutrophil apoptosis in very early rheumatoid arthritis patients is abrogated by methotrexate therapy.Smoking is a predictor of worse trabecular mechanical performance in hip fragility fracture patients.The skeletal phenotype of achondrogenesis type 1A is caused exclusively by cartilage defects.Clcn7F318L/+ as a new mouse model of Albers-Schönberg disease.Low osteocalcin/collagen type I bone gene expression ratio is associated with hip fragility fractures.Lymphotoxin-α 252 A>G polymorphism: a link between disease susceptibility and dyslipidemia in rheumatoid arthritis?Evaluation of bone mechanical strenght and fracture risk assessment (Frax) in patients with hip joint replacement surgery.Rheumatoid arthritis bone fragility is associated with upregulation of IL17 and DKK1 gene expression.Ankylosing spondylitis susceptibility and severity--contribution of TNF gene promoter polymorphisms at positions -238 and -308.A Method for the Evaluation of Femoral Head Trabecular Bone Compressive PropertiesMicro-computed tomography and compressive characterization of trabecular boneChronic arthritis directly induces quantitative and qualitative bone disturbances leading to compromised biomechanical propertiesAssessment of laboratory measurements and −308 TNFα gene promoter polymorphisms in normal bone mineral densityTNF promoter -308 G>A and LTA 252 A>G polymorphisms in Portuguese patients with systemic lupus erythematosusA role for G protein-coupled receptor 137b in bone remodeling in mouse and zebrafishcelsr1a is essential for tissue homeostasis and onset of aging phenotypes in the zebrafish
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description
hulumtuese
@sq
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Joana Caetano-Lopes
@ast
Joana Caetano-Lopes
@en
Joana Caetano-Lopes
@es
Joana Caetano-Lopes
@nl
Joana Caetano-Lopes
@sl
type
label
Joana Caetano-Lopes
@ast
Joana Caetano-Lopes
@en
Joana Caetano-Lopes
@es
Joana Caetano-Lopes
@nl
Joana Caetano-Lopes
@sl
prefLabel
Joana Caetano-Lopes
@ast
Joana Caetano-Lopes
@en
Joana Caetano-Lopes
@es
Joana Caetano-Lopes
@nl
Joana Caetano-Lopes
@sl
P108
P106
P1153
16300521000
P21
P31
P496
0000-0003-0310-4641