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A baboon model for endometriosis: implications for fertilityChanges in eutopic endometrial gene expression during the progression of experimental endometriosis in the baboon, Papio anubis.Induction of endometriosis alters the peripheral and endometrial regulatory T cell population in the non-human primate.Lesion kinetics in a non-human primate model of endometriosisInhibition of delta-like ligand 4 induces luteal hypervascularization followed by functional and structural luteolysis in the primate ovary.Connexin expression pattern in the endometrium of baboons is influenced by hormonal changes and the presence of endometriotic lesions.Soluble vascular endothelial growth factor receptor 1 inhibits edema and epithelial proliferation induced by 17beta-estradiol in the mouse uterus.Induced endometriosis in the baboon (Papio anubis) increases the expression of the proangiogenic factor CYR61 (CCN1) in eutopic and ectopic endometria.Altered expression of HOXA10 in endometriosis: potential role in decidualizationThe Altered Distribution of the Steroid Hormone Receptors and the Chaperone Immunophilin FKBP52 in a Baboon Model of Endometriosis Is Associated With Progesterone Resistance During the Window of Uterine ReceptivityThe Estrogen Early Response Gene FOS Is Altered in a Baboon Model of Endometriosis1In VivoInfusion of Interleukin-1β and Chorionic Gonadotropin Induces Endometrial Changes that Mimic Early Pregnancy Events in the BaboonExpression of vascular endothelial growth factor (VEGF)-D and its receptor, VEGF receptor 3, as a prognostic factor in endometrial carcinomaFuture Directions in Endometriosis Research
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description
hulumtuese
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researcher
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wetenschapper
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հետազոտող
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name
Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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Julie M Hastings
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P106
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0000-0002-0090-6924