Functional and histological evidence for the protective effect of NXY-059 in a primate model of stroke when given 4 hours after occlusion. - Test2 - elhamkhani - TriplyDB

Functional and histological evidence for the protective effect of NXY-059 in a primate model of stroke when given 4 hours after occlusion.

Functional and histological evidence for the protective effect of NXY-059 in a primate model of stroke when given 4 hours after occlusion.

http://www.wikidata.org/entity/Q44550693

about

Neuroprotection for ischemic stroke: past, present and future Effects of NXY-059 in experimental stroke: an individual animal meta-analysis Oxidative Stress and the Use of Antioxidants in Stroke Neuroprotection for stroke: current status and future perspectives Nonhuman primate models of stroke for translational neuroprotection research.Treating the acute stroke patient as an emergency: current practices and future opportunities.Update of the stroke therapy academic industry roundtable preclinical recommendations Nitrones as therapeutics.Intraluminal thread model of focal stroke in the non-human primate.Translational stroke research using a rabbit embolic stroke model: a correlative analysis hypothesis for novel therapy development Permanent or transient chronic ischemic stroke in the non-human primate: behavioral, neuroimaging, histological, and immunohistochemical investigations.Long-term neuroprotection from a potent redox-modulating metalloporphyrin in the rat.NXY-059: review of neuroprotective potential for acute stroke.A more consistent intraluminal rhesus monkey model of ischemic stroke.Climbing STAIRs towards clinical trials with a novel PARP-1 inhibitor for the treatment of ischemic stroke.Nitrones as therapeutics in age-related diseases.The use of the Chandler loop to examine the interaction potential of NXY-059 on the thrombolytic properties of rtPA on human thrombi in vitro Is NXY-059 an advancement in the treatment of acute ischaemic stroke?Clinical pharmacological issues in the development of acute stroke therapies.The past and future of neuroprotection in cerebral ischaemic stroke.Pharmacological approaches to acute ischaemic stroke: reperfusion certainly, neuroprotection possibly Endovascular ischemic stroke models of adult rhesus monkeys: a comparison of two endovascular methods Translating promising preclinical neuroprotective therapies to human stroke trials.Neuroprotection for ischaemic stroke: translation from the bench to the bedside.NADPH oxidases as therapeutic targets in ischemic stroke.Small molecule inhibitors in the treatment of cerebral ischemia.Ischemic stroke: experimental models and reality.The nitrone disodium 2,4-sulphophenyl-N-tert-butylnitrone is without cytoprotective effect on sodium nitroprusside-induced cell death in N1E-115 neuroblastoma cells in vitro.Neuroprotection by S-PBN in hyperglycemic ischemic brain injury in rats.New pathways for evaluating potential acute stroke therapies.How is edaravone effective against acute ischemic stroke and amyotrophic lateral sclerosis?Induction of the cell survival kinase Sgk1: A possible novel mechanism for α-phenyl-N-tert-butyl nitrone in experimental stroke.NXY-059 does not affect bleeding time in healthy volunteers: a randomized, double-blind, placebo-controlled, 3-period crossover phase I study.NXY-059 does not significantly interact with furosemide in healthy volunteers.Permanent middle cerebral artery occlusion in sheep: a novel large animal model of focal cerebral ischemia.Targeting NMDA receptors in stroke: new hope in neuroprotection.Refocusing Neuroprotection in Cerebral Reperfusion Era: New Challenges and Strategies.The Biochemical Basis of Hydroxymethylglutaryl-CoA Reductase Inhibitors as Neuroprotective Agents in Aneurysmal Subarachnoid Hemorrhage.Neuroprotection: where to now?

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P2860

Functional and histological evidence for the protective effect of NXY-059 in a primate model of stroke when given 4 hours after occlusion.

http://www.wikidata.org/entity/Q44550693

description

2003 nî lūn-bûn

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Functional and histological ev ...... given 4 hours after occlusion.

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Jonathan W B Marshall

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