Inhibition of cyclooxygenase-2 improves cardiac function after myocardial infarction in the mouse.
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Prostaglandin E Receptor Subtype 4 Signaling in the Heart: Role in Ischemia/Reperfusion Injury and Cardiac HypertrophyThe human subject: an integrative animal model for 21(st) century heart failure researchThe contribution of prostaglandins versus prostacyclin in ventricular remodeling during heart failureDownregulation of NADPH oxidase, antioxidant enzymes, and inflammatory markers in the heart of streptozotocin-induced diabetic rats by N-acetyl-L-cysteinePKA, Rap1, ERK1/2, and p90RSK mediate PGE2 and EP4 signaling in neonatal ventricular myocytes.Cyclooxygenase-2 inhibition provides lasting protection against neonatal hypoxic-ischemic brain injury.Preserved heart function and maintained response to cardiac stresses in a genetic model of cardiomyocyte-targeted deficiency of cyclooxygenase-2.MAPK-activated protein kinase-2 in cardiac hypertrophy and cyclooxygenase-2 regulation in heart.Lack of microsomal prostaglandin E synthase-1 reduces cardiac function following angiotensin II infusionProstaglandin E2 increases cardiac fibroblast proliferation and increases cyclin D expression via EP1 receptor.Cyclooxygenase-2 signaling in vocal fold fibroblastsCardiac thromboxane A2 receptor activation does not directly induce cardiomyocyte hypertrophy but does cause cell death that is prevented with gentamicin and 2-APB.Infiltration of inflammatory cells plays an important role in matrix metalloproteinase expression and activation in the heart during sepsis.Substance P in heart failure: the good and the bad.Tryptase activates isolated adult cardiac fibroblasts via protease activated receptor-2 (PAR-2).Angiotensin-(1-7) abrogates mitogen-stimulated proliferation of cardiac fibroblasts.Cyclooxygenase in normal human tissues--is COX-1 really a constitutive isoform, and COX-2 an inducible isoform?Effect of DanQi Pill on PPARα, lipid disorders and arachidonic acid pathway in rat model of coronary heart disease.Ischemic preconditioning via epsilon protein kinase C activation requires cyclooxygenase-2 activation in vitroRole of COX-2-derived PGE2 on vascular stiffness and function in hypertension.Selective cyclooxygenase-2 inhibition protects against myocardial damage in experimental acute ischemiaMorphological and molecular changes of the myocardium after left ventricular mechanical supportPathophysiology of cyclooxygenases in cardiovascular homeostasis.Opposite Modulatory Effects of Selective and Non-Selective Cyclooxygenase Inhibition on Cardiovascular and Autonomic Consequences of Cyclosporine in Female Rats.Protective mechanisms of NO preconditioning against NO-induced apoptosis in H9c2 cells: role of PKC and COX-2.A Model of Left Ventricular Dysfunction Complicated by CAWS Arteritis in DBA/2 Mice.Augmented oxidative stress and preserved vasoconstriction induced by hydrogen peroxide in coronary arteries in obesity: role of COX-2.Reduced cardiac remodeling and function in cardiac-specific EP4 receptor knockout mice with myocardial infarctionThe effects of cyclooxygenase and nitric oxide synthase inhibition on cardiodynamic parameters and coronary flow in isolated rat hearts.Regulation of the membrane-localized prostaglandin E synthases mPGES-1 and mPGES-2 in cardiac myocytes and fibroblasts.Overexpression of prostaglandin EP3 receptors activates calcineurin and promotes hypertrophy in the murine heart.Effect of selective and non-selective cyclooxygenase inhibitors on doxorubicin-induced cardiotoxicity and nephrotoxicity in rats.The PGE2-Stat3 interaction in doxorubicin-induced myocardial apoptosis.PGE2 stimulates human brain natriuretic peptide expression via EP4 and p42/44 MAPK.Anti-inflammatory therapies in myocardial infarction: failures, hopes, and challenges.Overexpression of prostaglandin E2 EP4 receptor improves cardiac function after myocardial infarction.Disruption of COX-2 modulates gene expression and the cardiac injury response to doxorubicin.Low-dose celecoxib improves coronary function after acute myocardial ischaemia in rabbits.
P2860
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P2860
Inhibition of cyclooxygenase-2 improves cardiac function after myocardial infarction in the mouse.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@en
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@nl
type
label
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@en
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@nl
prefLabel
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@en
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@nl
P2093
P2860
P1476
Inhibition of cyclooxygenase-2 ...... rdial infarction in the mouse.
@en
P2093
Alicia Leung
Margot C LaPointe
Mariela Mendez
Xiao-Ping Yang
Zhenyin Tao
P2860
P304
P356
10.1152/AJPHEART.00136.2003
P577
2003-12-11T00:00:00Z