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Comparative cardiac toxicity of anthracyclines in vitro and in vivo in the mouseEnhanced interleukin-1 activity contributes to exercise intolerance in patients with systolic heart failureRight ventricular dysfunction following acute myocardial infarction in the absence of pulmonary hypertension in the mouse.Alterations in the interleukin-1/interleukin-1 receptor antagonist balance modulate cardiac remodeling following myocardial infarction in the mouseAltered oxido-reductive state in the diabetic heart: loss of cardioprotection due to protein disulfide isomeraseThe inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse.A mouse model of heart failure with preserved ejection fraction due to chronic infusion of a low subpressor dose of angiotensin II.A mouse model of radiation-induced cardiomyopathy.The serine protease HtrA1 specifically interacts and degrades the tuberous sclerosis complex 2 protein.Pharmacologic inhibition of phosphoinositide 3-kinase gamma (PI3Kγ) promotes infarct resorption and prevents adverse cardiac remodeling after myocardial infarction in mice.Galectin-1 controls cardiac inflammation and ventricular remodeling during acute myocardial infarction.Anakinra in experimental acute myocardial infarction--does dosage or duration of treatment matter?Apoptosis in patients with acute myocarditis.Interleukin-1 trap attenuates cardiac remodeling after experimental acute myocardial infarction in mice.Pharmacologic inhibition of myeloid differentiation factor 88 (MyD88) prevents left ventricular dilation and hypertrophy after experimental acute myocardial infarction in the mouse.Interleukin-1beta modulation using a genetically engineered antibody prevents adverse cardiac remodelling following acute myocardial infarction in the mouse.The role of PDI as a survival factor in cardiomyocyte ischemia.Suppression of histone deacetylases worsens right ventricular dysfunction after pulmonary artery banding in rats.Alpha-1 antitrypsin inhibits caspase-1 and protects from acute myocardial ischemia-reperfusion injury.Recombinant human interleukin-1 receptor antagonist provides cardioprotection during myocardial ischemia reperfusion in the mouse.GS-6201, a selective blocker of the A2B adenosine receptor, attenuates cardiac remodeling after acute myocardial infarction in the mouse.Interleukin-1β blockade improves cardiac remodelling after myocardial infarction without interrupting the inflammasome in the mouse.Phosphodiesterase-5 inhibition and cardioprotection: potential role of hydrogen sulfide.Interleukin-1β induces a reversible cardiomyopathy in the mouse.Right ventricular systolic dysfunction in patients with reperfused ST-segment elevation acute myocardial infarctionInflammasome, pyroptosis, and cytokines in myocardial ischemia-reperfusion injury
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description
hulumtues
@sq
researcher
@en
wetenschapper
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հետազոտող
@hy
name
Stefano Toldo
@ast
Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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prefLabel
Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
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Stefano Toldo
@sl
P106
P1153
35729889100
P21
P31
P496
0000-0002-1588-4019