Pivotal Advance: Invariant NKT cells reduce accumulation of inflammatory monocytes in the lungs and decrease immune-pathology during severe influenza A virus infection.
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Universal immunity to influenza must outwit immune evasionRole of non-conventional T lymphocytes in respiratory infections: the case of the pneumococcusCoadministration of Hedera helix L. Extract Enabled Mice to Overcome Insufficient Protection against Influenza A/PR/8 Virus Infection under Suboptimal Treatment with OseltamivirInnate immune sensing and response to influenza.α-Galactosylceramide protects swine against influenza infection when administered as a vaccine adjuvant.Rapid control of pandemic H1N1 influenza by targeting NKT-cells.Role of T cell immunity in recovery from influenza virus infectionPseudotyped influenza A virus as a vaccine for the induction of heterotypic immunityHigh levels of virus-specific CD4+ T cells predict severe pandemic influenza A virus infection.Invariant NKT cell response to dengue virus infection in human.Inflammatory mechanisms in sepsis: elevated invariant natural killer T-cell numbers in mouse and their modulatory effect on macrophage function.Invariant NKT cells: regulation and function during viral infection.Determinants of host susceptibility to murine respiratory syncytial virus (RSV) disease identify a role for the innate immunity scavenger receptor MARCO gene in human infantsInterleukin-22 is produced by invariant natural killer T lymphocytes during influenza A virus infection: potential role in protection against lung epithelial damagesA20 Deficiency in Lung Epithelial Cells Protects against Influenza A Virus InfectionAbnormalities in iNKT cells are associated with impaired ability of monocytes to produce IL-10 and suppress T-cell proliferation in sarcoidosis.Natural killer T cells contribute to the control of acute retroviral infection.M1-like monocytes are a major immunological determinant of severity in previously healthy adults with life-threatening influenza.C-C Motif Chemokine Ligand 2 (CCL2) Mediates Acute Lung Injury Induced by Lethal Influenza H7N9 Virus.Role of type 1 natural killer T cells in pulmonary immunity.The amazing innate immune response to influenza A virus infection.Regulation of NKT Cell Localization in Homeostasis and InfectionInfluenza A virus-induced release of interleukin-10 inhibits the anti-microbial activities of invariant natural killer T cells during invasive pneumococcal superinfection.Protective role of LGP2 in influenza virus pathogenesis.Natural Killer T Cells: An Ecological Evolutionary Developmental Biology Perspective.Epithelial-derived TGF-β1 acts as a pro-viral factor in the lung during influenza A infection.CD1-Restricted T Cells During Persistent Virus Infections: "Sympathy for the Devil".Age-related M1/M2 phenotype changes in circulating monocytes from healthy/unhealthy individualsNatural Killer T Cells and Mucosal-Associated Invariant T Cells in Lung InfectionsLipid-Reactive T Cells in Immunological Disorders of the LungThe Pathophysiological Relevance of the iNKT Cell/Mononuclear Phagocyte Crosstalk in TissuesMAIT cells contribute to protection against lethal influenza infection in vivo
P2860
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P2860
Pivotal Advance: Invariant NKT cells reduce accumulation of inflammatory monocytes in the lungs and decrease immune-pathology during severe influenza A virus infection.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@en
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@nl
type
label
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@en
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@nl
prefLabel
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@en
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@nl
P2093
P2860
P356
P1476
Pivotal Advance: Invariant NKT ...... e influenza A virus infection.
@en
P2093
Andrew J McMichael
Colin Clelland
Kambez Benam
Laura Denney
Ling-Pei Ho
Suzanne Cole
Wai Ling Kok
P2860
P304
P356
10.1189/JLB.0411184
P577
2011-10-14T00:00:00Z