Regulation of the interferon-alpha production induced by RNA-containing immune complexes in plasmacytoid dendritic cells. - Test2 - elhamkhani - TriplyDB

Regulation of the interferon-alpha production induced by RNA-containing immune complexes in plasmacytoid dendritic cells.

Regulation of the interferon-alpha production induced by RNA-containing immune complexes in plasmacytoid dendritic cells.

http://www.wikidata.org/entity/Q45915295

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Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic Cells The Role of Damage-Associated Molecular Patterns in Human Diseases: Part I - Promoting inflammation and immunity Cause and consequences of the activated type I interferon system in SLE Anti-U1 RNP antibodies in cerebrospinal fluid are associated with central neuropsychiatric manifestations in systemic lupus erythematosus and mixed connective tissue disease The dendritic cell response to classic, emerging, and homeostatic danger signals. Implications for autoimmunity Dendritic cells in systemic lupus erythematosus TLR tolerance reduces IFN-alpha production despite plasmacytoid dendritic cell expansion and anti-nuclear antibodies in NZB bicongenic mice.C1q deficiency leads to the defective suppression of IFN-alpha in response to nucleoprotein containing immune complexes.Antigen is required for maturation and activation of pathogenic anti-DNA antibodies and systemic inflammation.Type I interferon-mediated skewing of the serotonin synthesis is associated with severe disease in systemic lupus erythematosus The type I interferon system in the etiopathogenesis of autoimmune diseases.Type I interferon and systemic lupus erythematosus.Plasmacytoid dendritic cells in atherosclerosis.Toll-like receptors as therapeutic targets for autoimmune connective tissue diseases The complement receptor 3 (CD11b/CD18) agonist Leukadherin-1 suppresses human innate inflammatory signalling.Fueling autoimmunity: type I interferon in autoimmune diseases.Potential role of IFNα in adult lupus.Targeting B cells for the treatment of SLE: the beginning of the end or the end of the beginning?Toll-like receptors in rheumatic diseases: are we paying a high price for our defense against bugs?Disease mechanisms in rheumatology--tools and pathways: plasmacytoid dendritic cells and their role in autoimmune rheumatic diseases.Aberrant B-lymphocyte responses in lupus: inherent or induced and potential therapeutic targets.B-lymphocyte signalling abnormalities and lupus immunopathology.Immune mechanisms in polymyositis and dermatomyositis and potential targets for therapy.Innate immunity in systemic sclerosis pathogenesis.Plasmacytoid Dendritic Cells: Neglected Regulators of the Immune Response to Staphylococcus aureus.Systemic Lupus Erythematosus--A Disease with A Dysregulated Type I Interferon System.Ncf1 polymorphism reveals oxidative regulation of autoimmune chronic inflammation.Antinuclear antibody testing - misunderstood or misbegotten?FcγRIIB prevents inflammatory type I IFN production from plasmacytoid dendritic cells during a viral memory response.Positivity for anti-RNP antibody is a risk factor for adverse effects caused by trimethoprim-sulfamethoxazole, a prophylactic agent for P. jiroveci pneumonia, in patients with connective tissue diseases.Netting neutrophils are major inducers of type I IFN production in pediatric systemic lupus erythematosus.IFN-α production by plasmacytoid dendritic cell associations with polymorphisms in gene loci related to autoimmune and inflammatory diseases.Toll-like receptor-mediated, enhanced production of profibrotic TIMP-1 in monocytes from patients with systemic sclerosis: role of serum factors.New Perspectives in Rheumatology: Biomarkers as Entry Criteria for Clinical Trials of New Therapies for Systemic Lupus Erythematosus: The Example of Antinuclear Antibodies and Anti-DNA.Blood-Borne RNA Correlates with Disease Activity and IFN-Stimulated Gene Expression in Systemic Lupus Erythematosus.Autologous tolerogenic dendritic cells derived from monocytes of systemic lupus erythematosus patients and healthy donors show a stable and immunosuppressive phenotype.Type I interferon-mediated autoimmune diseases: pathogenesis, diagnosis and targeted therapy.Activated T cells enhance interferon-α production by plasmacytoid dendritic cells stimulated with RNA-containing immune complexes.Circulating microRNA expression profiles associated with systemic lupus erythematosus.Report of the inaugural Interferon Research Summit: interferon in inflammatory diseases.

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P2860

Regulation of the interferon-alpha production induced by RNA-containing immune complexes in plasmacytoid dendritic cells.

http://www.wikidata.org/entity/Q45915295

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Regulation of the interferon-a ...... plasmacytoid dendritic cells.

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Regulation of the interferon-a ...... plasmacytoid dendritic cells.

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type

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Regulation of the interferon-a ...... plasmacytoid dendritic cells.

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Regulation of the interferon-a ...... plasmacytoid dendritic cells.

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prefLabel

Regulation of the interferon-a ...... plasmacytoid dendritic cells.

@en

Regulation of the interferon-a ...... plasmacytoid dendritic cells.

@nl

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Arthritis & Rheumatology

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Regulation of the interferon-a ...... plasmacytoid dendritic cells.

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Doreen Finke

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Gunnar V Alm

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Johan Rönnelid

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Lars Rönnblom

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Linda Mathsson

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Maija-Leena Eloranta

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Tanja Lövgren

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P2860

Human lupus autoantibody-DNA complexes activate DCs through cooperation of CD32 and TLR9.

The combination of apoptotic U937 cells and lupus IgG is a potent IFN-alpha inducer.

Prostaglandin E2 suppressed IL-15-mediated human NK cell function through down-regulation of common gamma-chain.

The reciprocal interaction of NK cells with plasmacytoid or myeloid dendritic cells profoundly affects innate resistance functions.

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2418-2427

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scholarly article

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10.1002/ART.24686

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8

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60

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Berthold Kastner

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2009-08-01T00:00:00Z

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26707463

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19644885

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