Inflammation in bone physiology and pathology. - Test2 - elhamkhani - TriplyDB

Inflammation in bone physiology and pathology.

Inflammation in bone physiology and pathology.

http://www.wikidata.org/entity/Q46010612

about

Inflammation in bone physiology and pathology.

http://www.wikidata.org/entity/Q46010612

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Inflammation in bone physiology and pathology.

@en

Inflammation in bone physiology and pathology.

@nl

type

label

Inflammation in bone physiology and pathology.

@en

Inflammation in bone physiology and pathology.

@nl

prefLabel

Inflammation in bone physiology and pathology.

@en

Inflammation in bone physiology and pathology.

@nl

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Current Opinion in Rheumatology

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Inflammation in bone physiology and pathology

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Iannis E Adamopoulos

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Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva

Role of gamma-delta (γδ) T cells in autoimmunity

Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation

Signal adaptor DAP10 associates with MDL-1 and triggers osteoclastogenesis in cooperation with DAP12

Genetic regulation of osteoclast development and function

Evolving concepts of rheumatoid arthritis

SH3BP2 cherubism mutation potentiates TNF-α-induced osteoclastogenesis via NFATc1 and TNF-α-mediated inflammatory bone loss.

NFATc1 in mice represses osteoprotegerin during osteoclastogenesis and dissociates systemic osteopenia from inflammation in cherubism.

Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interaction

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59-64

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scholarly article

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10.1097/BOR.0000000000000449

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1

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30

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Iannis E Adamopoulos

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2018-01-01T00:00:00Z

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29016371

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5963529

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