Enhanced sleep reverses memory deficits and underlying pathology in Drosophila models of Alzheimer's disease.

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Identification of Genes that Maintain Behavioral and Structural Plasticity during Sleep Loss.β-Amyloid accumulation in the human brain after one night of sleep deprivation.

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Enhanced sleep reverses memory deficits and underlying pathology in Drosophila models of Alzheimer's disease.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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Enhanced sleep reverses memory ...... models of Alzheimer's disease.

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Enhanced sleep reverses memory ...... models of Alzheimer's disease.

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Enhanced sleep reverses memory ...... models of Alzheimer's disease.

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Enhanced sleep reverses memory ...... models of Alzheimer's disease.

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Enhanced sleep reverses memory ...... models of Alzheimer's disease.

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Bruno van Swinderen

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Denis English

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Jeff Donlea

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Lijuan Cao

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Markus Klose

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Raphaelle Winsky-Sommerer

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Stephane Dissel

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Dissecting the pathological effects of human Abeta40 and Abeta42 in Drosophila: a potential model for Alzheimer's disease

Amyloid-beta dynamics are regulated by orexin and the sleep-wake cycle

Sleep, Performance, and Memory in Flies

Sleep, circadian rhythms, and the pathogenesis of Alzheimer disease

Sleep Facilitates Memory by Blocking Dopamine Neuron-Mediated Forgetting

Targeted gene expression as a means of altering cell fates and generating dominant phenotypes

Modeling the complex pathology of Alzheimer's disease in Drosophila

Circadian remodeling of neuronal circuits involved in rhythmic behavior

Characterization of a Drosophila Alzheimer's disease model: pharmacological rescue of cognitive defects

Sleep function and synaptic homeostasis

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15-26

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scholarly article

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10.1016/J.NBSCR.2016.09.001

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Raphaelle Winsky-Sommerer

Bruno van Swinderen

Jeff Donlea

Paul J. Shaw

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2016-09-28T00:00:00Z

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29094110

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Alzheimer's disease

Drosophila

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Enhanced sleep reverses memory deficits and underlying pathology in Drosophila models of Alzheimer's disease.

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Enhanced sleep reverses memory deficits and underlying pathology in Drosophila models of Alzheimer's disease.

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