about
The evolution and pathology of frontotemporal dementiaThe new mutation, E46K, of alpha-synuclein causes Parkinson and Lewy body dementiaCommon variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions.High-resolution whole-genome analysis of skull base chordomas implicates FHIT loss in chordoma pathogenesisStructural features of three ureterocele calculi.Malignant ganglioglioma: case report and review of literature.Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar DegenerationClinical and pathological overlap between frontotemporal dementia, primary progressive aphasia and corticobasal degeneration: the Pick complex.FUS pathology defines the majority of tau- and TDP-43-negative frontotemporal lobar degeneration.FET proteins TAF15 and EWS are selective markers that distinguish FTLD with FUS pathology from amyotrophic lateral sclerosis with FUS mutations.The neuropathology and biochemistry of frontotemporal dementia.Systematic review of primary intracranial glioblastoma multiforme with symptomatic spinal metastases, with two illustrative patients.Relationship between frontotemporal dementia and corticobasal degeneration/progressive supranuclear palsy.Rate of progression of cognitive decline in Alzheimer's disease: effect of butyrylcholinesterase K gene variationA multigenerational pedigree of late-onset Alzheimer's disease implies new genetic causes.Clinical and immunologic features of an atypical intracranial mycobacterium avium complex (MAC) infection compared with those of pulmonary MAC infectionsNeuropathology of hereditary forms of frontotemporal dementia and parkinsonism.A short illustrated review of sellar region schwannomas.The diagnosis and course of frontotemporal dementia.Primary progressive aphasia: diagnosis, varieties, evolution.Amygdaloid sclerosis in temporal lobe epilepsy.Cerebrovascular pathology in Alzheimer's disease: cause, effect or epiphenomenon?Do alpha-synuclein aggregates in autonomic plexuses predate Lewy body disorders?: a cohort study.A TAP2 genotype associated with Alzheimer's disease in APOE4 carriers.Chromogranin A-like immunoreactivity in the human brain: distribution in bulbar medulla and cerebral cortex.Amusia as an early manifestation of frontotemporal dementia caused by a novel progranulin mutation.Transportin 1 accumulates specifically with FET proteins but no other transportin cargos in FTLD-FUS and is absent in FUS inclusions in ALS with FUS mutations.Spinal epidural cavernous hemangioma in an HIV-positive patient.MPTP induces intranuclear rodlet formation in midbrain dopaminergic neurons.Indices of resective surgery effectiveness for intractable nonlesional focal epilepsy.Behavioral quantitation is more sensitive than cognitive testing in frontotemporal dementia.A 63 year old woman with white matter lesions and pachymeningeal inflammation.Correlation between MRI and clinico-pathological manifestations in Lewis rats protected from experimental allergic encephalomyelitis by acylated synthetic peptide of myelin basic protein.Hemorrhagic epidermoid cyst in a patient with generalized tonic clonic seizure.Rosai-Dorfman disease involving the neurohypophysis.Preferred terminology.Frontotemporal degeneration, Pick's disease, Pick complex, and Ravel.Chromogranin A inhibits dopamine release from rat striatal slices.Promyelocytic leukaemia-immunoreactive neuronal intranuclear rodlets in the human brain.Chromogranin A, a soluble synaptic vesicle protein, is found in cortical neurons other than previously defined peptidergic neurons in the human neocortex.
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description
hulumtues
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researcher
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հետազոտող
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name
David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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David G. Munoz
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