Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning. - Test2 - elhamkhani - TriplyDB

Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.

Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.

http://www.wikidata.org/entity/Q46460215

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Interaction of Hydrogen Sulfide with Nitric Oxide in the Cardiovascular System Hydrogen sulfide chemical biology: pathophysiological roles and detection The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning A tale of two gases: NO and H2S, foes or friends for life?Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice.The therapeutic potential of antioxidants, ER chaperones, NO and H2S donors, and statins for treatment of preeclampsia.mTORC2 phosphorylation of Akt1: a possible mechanism for hydrogen sulfide-induced cardioprotection.Novel insights into hydrogen sulfide--mediated cytoprotection.Hydrogen sulfide in cell survival: a double-edged sword.Protective effect of hydrogen sulphide against 6-OHDA-induced cell injury in SH-SY5Y cells involves PKC/PI3K/Akt pathway Sodium Hydrosulphide alleviates remote lung injury following limb traumatic injury in rats.Hydrogen sulfide mitigates transition from compensatory hypertrophy to heart failure Cardioprotective effects of hydrogen sulfide Hydrogen sulfide improves cardiomyocytes electrical remodeling post ischemia/reperfusion injury in rats Mediation of exogenous hydrogen sulfide in recovery of ischemic post-conditioning-induced cardioprotection via down-regulating oxidative stress and up-regulating PI3K/Akt/GSK-3β pathway in isolated aging rat hearts Cardioprotection by H2S engages a cGMP-dependent protein kinase G/phospholamban pathway.Hydrogen sulfide is an endogenous potentiator of T cell activation.Exogenous hydrogen sulfide restores cardioprotection of ischemic post-conditioning via inhibition of mPTP opening in the aging cardiomyocytes.Hydrogen sulfide in biochemistry and medicine.Endogenous hydrogen sulfide mediates the cardioprotection induced by ischemic postconditioning in the early reperfusion phase.Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.Garlic oil polysulfides: H2S- and O2-independent prooxidants in buffer and antioxidants in cells.The hydrogen sulfide donor, GYY4137, exhibits anti-atherosclerotic activity in high fat fed apolipoprotein E(-/-) mice.Hydrogen sulfide and the vasculature: a novel vasculoprotective entity and regulator of nitric oxide bioavailability?Hydrogen sulfide regulates Ca(2+) homeostasis mediated by concomitantly produced nitric oxide via a novel synergistic pathway in exocrine pancreas.Alternative Interventions to Prevent Oxidative Damage following Ischemia/Reperfusion The New Synthetic H2S-Releasing SDSS Protects MC3T3-E1 Osteoblasts against H2O2-Induced Apoptosis by Suppressing Oxidative Stress, Inhibiting MAPKs, and Activating the PI3K/Akt Pathway.Endogenous production of H2S in the gastrointestinal tract: still in search of a physiologic function.Hydrogen sulfide-mediated myocardial pre- and post-conditioning.Treatment of Myocardial Ischemia/Reperfusion Injury by Ischemic and Pharmacological Postconditioning.Beyond a Gasotransmitter: Hydrogen Sulfide and Polysulfide in Cardiovascular Health and Immune Response.Interplay of hydrogen sulfide and nitric oxide on the pacemaker activity of interstitial cells of cajal from mouse small intestine.Hydrogen sulphide protects mouse pancreatic β-cells from cell death induced by oxidative stress, but not by endoplasmic reticulum stress.Hydrogen sulphide induces micro opioid receptor-dependent analgesia in a rodent model of visceral pain.H2S mitigates severe acute pancreatitis through the PI3K/AKT-NF-κB pathway in vivo Hydrogen sulfide increases nitric oxide production from endothelial cells by an akt-dependent mechanism.H2S regulation of nitric oxide metabolism.Hydrogen sulfide mediates the cardioprotective effects of gene therapy with PKG-Iα.The effects of hydrogen sulfide under sevoflurane administration against ischemia and reperfusion injury in isolated rat heart.Hydrogen sulfide interacts with nitric oxide in the heart: possible involvement of nitroxyl.

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P2860

Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.

http://www.wikidata.org/entity/Q46460215

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2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年學術文章

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... by ischemic postconditioning.

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Endogenous hydrogen sulphide m ...... d by ischemic postconditioning

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Chun Shin Foo

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Kay Li Neo

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Qian Chen Yong

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Shiau Wei Lee

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Xin Chen

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P2860

The noble gas xenon induces pharmacological preconditioning in the rat heart in vivo via induction of PKC-epsilon and p38 MAPK

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

The vasorelaxant effect of H(2)S as a novel endogenous gaseous K(ATP) channel opener

Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

Two's company, three's a crowd: can H2S be the third endogenous gaseous transmitter?

Localization, anchoring, and functions of protein kinase C isozymes in the heart.

Infarct-sparing effect of myocardial postconditioning is dependent on protein kinase C signalling.

Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade.

Postconditioning--A new link in nature's armor against myocardial ischemia-reperfusion injury.

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