Neuronal responses to transient hypoglycaemia in the dorsal vagal complex of the rat brainstem.
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Sweet talk in the brain: glucosensing, neural networks, and hypoglycemic counterregulationInsights into the role of neuronal glucokinaseCentral nervous system control of gastrointestinal motility and secretion and modulation of gastrointestinal functions.2. Hypoglycemia Detection.Quantitative RT-PCR and immunoblot analyses reveal acclimated A2 noradrenergic neuron substrate fuel transporter, glucokinase, phospho-AMPK, and dopamine-β-hydroxylase responses to hypoglycemia.cAMP-dependent insulin modulation of synaptic inhibition in neurons of the dorsal motor nucleus of the vagus is altered in diabetic mice.NPY and VGF immunoreactivity increased in the arcuate nucleus, but decreased in the nucleus of the Tractus Solitarius, of type-II diabetic patients.Neuroendocrine responses to hypoglycemia.Glucose stimulation of hypothalamic MCH neurons involves K(ATP) channels, is modulated by UCP2, and regulates peripheral glucose homeostasis.Cellular and molecular cues of glucose sensing in the rat olfactory bulb.A novel slice preparation to study medullary oromotor and autonomic circuits in vitro.Role of dorsal vagal complex A2 noradrenergic neurons in hindbrain glucoprivic inhibition of the luteinizing hormone surge in the steroid-primed ovariectomized female rat: effects of 5-thioglucose on A2 functional biomarker and AMPK activityEstrogen regulates energy metabolic pathway and upstream adenosine 5'-monophosphate-activated protein kinase and phosphatase enzyme expression in dorsal vagal complex metabolosensory neurons during glucostasis and hypoglycemia.Caudal fourth ventricular administration of the AMPK activator 5-aminoimidazole-4-carboxamide-riboside regulates glucose and counterregulatory hormone profiles, dorsal vagal complex metabolosensory neuron function, and hypothalamic Fos expression.Enhanced NMDA receptor-mediated modulation of excitatory neurotransmission in the dorsal vagal complex of streptozotocin-treated, chronically hyperglycemic mice.CCK stimulation of GLP-1 neurons involves α1-adrenoceptor-mediated increase in glutamatergic synaptic inputsGastric relaxation induced by hyperglycemia is mediated by vagal afferent pathways in the rat.Molecular and functional changes in glucokinase expression in the brainstem dorsal vagal complex in a murine model of type 1 diabetesMinireview: The value of looking backward: the essential role of the hindbrain in counterregulatory responses to glucose deficit.Insulin reduces excitation in gastric-related neurons of the dorsal motor nucleus of the vagus.Glucose sensing by GABAergic neurons in the mouse nucleus tractus solitarii.Fasting-induced suppression of LH secretion does not require activation of ATP-sensitive potassium channels.Small decrements in systemic glucose provoke increases in hypothalamic blood flow prior to the release of counterregulatory hormonesRapid inhibition of neurons in the dorsal motor nucleus of the vagus by leptin.Dissociation between sensing and metabolism of glucose in sugar sensing neurones.Modulation of gastrointestinal vagal neurocircuits by hyperglycemia.Reduced GABAergic inhibition of kidney-related PVN neurons in streptozotocin-treated type 1 diabetic mouse.Plasticity of central autonomic neural circuits in diabetes.Astrocytes in the nucleus of the solitary tract are activated by low glucose or glucoprivation: evidence for glial involvement in glucose homeostasis.Glucose-induced inhibition: how many ionic mechanisms?Stimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons.Plasticity of vagal brainstem circuits in the control of gastrointestinal function.Astrocytes as brain interoceptors.Neural pathways that control the glucose counterregulatory response.Hypothalamic glucose sensing: making ends meet.Functional plasticity of central TRPV1 receptors in brainstem dorsal vagal complex circuits of streptozotocin-treated hyperglycemic miceGlycemic state regulates melanocortin, but not nesfatin-1, responsiveness of glucose-sensing neurons in the nucleus of the solitary tract.Nutrient Sensing: Another Chemosensitivity of the Olfactory System.Glycolysis selectively shapes the presynaptic action potential waveform.High glucose increases action potential firing of catecholamine neurons in the nucleus of the solitary tract by increasing spontaneous glutamate inputs.
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P2860
Neuronal responses to transient hypoglycaemia in the dorsal vagal complex of the rat brainstem.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
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@zh-hant
name
Neuronal responses to transien ...... complex of the rat brainstem.
@en
Neuronal responses to transien ...... complex of the rat brainstem.
@nl
type
label
Neuronal responses to transien ...... complex of the rat brainstem.
@en
Neuronal responses to transien ...... complex of the rat brainstem.
@nl
prefLabel
Neuronal responses to transien ...... complex of the rat brainstem.
@en
Neuronal responses to transien ...... complex of the rat brainstem.
@nl
P2860
P1476
Neuronal responses to transien ...... complex of the rat brainstem.
@en
P2093
Ann Maria Kruse Hansen
Robert H Balfour
P2860
P304
P356
10.1113/JPHYSIOL.2005.098822
P407
P50
P577
2005-11-10T00:00:00Z